1[1]Tator CH,Fehling MG.Review of the secondary injury theory mechanisms.J Neurosurg,1991,75 (1):15-26.
2[2]Bracken MB,Shepard MJ,Collins WF,et al.Methylprednisolone or naloxone treatment after acute spinal cord injury:1-year follow-up data.J Neurosurg,1992 ;76 (1):23-31.
3[3]Geisler FH,Dorsey FC,Coleman WP.Past and current clinical studies with GM-1 gangliasides in acute spinal cord injury.Ann Emerg Med,1993 ;22 (6):1041-1047.
4[4]Bracken MB,Holford TR,Effects of timing of methylprednisoloue of naloxone administration on recovery of segmental and long-tract neurological function I NASCI2.J Neurosurg,1993 ;79 (4):500-507.
5[5]Gentile NT,McIntosh TK.Antagonists of excitatory aminoacids and endogenous opioid peptides in the treatment of experimental central nervous system injury.Ann Emerg Med,1993 ;22 (6):1028-1034.
6[6]Yanase M,Sakou T,Fukuda T.Role of N-methyl-D-aspartate receptor in acute spinal cord injury.J Neurosurg,1995;83 (5):884-888.
9[9]Fernandez E,Pallni R,Lauretti,et al.Spinal cord transection in adult rats:effects of local infusion of nerve growth factor on the corticaspinal tract axons.Neurosurgery,1993,37:889-893.
10[10]Jakeman LB,Wei P,Guan I,et al,Related articles brain derived neurotrophic factor stimulates hindlimb stepping and sprouting of cholinergic fibers after spinal cord injury.Exp Neurol,1998,154:170-184.
