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危重症患者的内皮功能异常 被引量:6

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摘要 危重症患者的多器官衰竭及病死率很高,其机制可能与伴随的应激性高血糖、氧化应激及胰岛素抵抗引起血管内皮功能异常相关。强化胰岛素治疗可减少内皮激活、抑制血管内皮损伤、细胞缺氧以及抗炎等,从而减少器官衰竭发生率及病死率。本文拟对危重症患者内皮功能异常的发生机制进行综述。
作者 邹森彪
机构地区 青海大学医学院
出处 《牡丹江医学院学报》 2008年第3期83-86,共4页 Journal of Mudanjiang Medical University
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