3[1]Kortea S M,Koolhaasb J M,Wingfieldc J C.The Darwinian concept of stress:benefits of allostasis and costs of allostatic load and the trade-offs in health and disease.Neuroscience and Biobehavioral Reviews,2005,29:3-38.
4[2]Dunn A.Nervous system-immune system interaction:an overview.Journal of Receptor Research,1988,8:589-607.
5[3]Moberg G P,Mench J A.The Biology of Animal Stress.CAB International Press,368.1-21.
6[4]Sarah C C,Mary P.Animal models of CRH excess and CRH receptor deficiency display altered adaptations to stress.Peptides,2001,22:733-741.
7[5]De Kloet E.Brain corticosteroid receptor balance and homeostatic control.Front euroendocrinol,1991,12:95-164.
8[6]DeRijk R,Michelson D,Karp B.Exercise and circadian rhythm-induced variations in plasma cortisol differentially regulate interleukin-1 (IL-lbeta),IL-6,and tumor necrosis factor-alpha (TNF alpha),production in humans:high sensitivity of TNF alpha and resistance of IL-6.Journal of Clinic Endocrine Metabolism,1997,82:2182-2191.
9[7]Chikanza I C,Grossman A B.Neuroendocrine immune responses in inflammation:the concept of the neuroendocrine immune loop.Baillieres Clinic Rheumato,1996,10:199-225.
10[8]Elsasser T H,Sartin J L,McMahon C,Romo G,Fayer R,Blagburn B.Changes in somatotropic axis response and body composition during growth hormone administration in progressive cachectic parasitism.Domestic Animal Endocrinology,1998,15:239-255.
