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胰岛β细胞瘦素抵抗在高胰岛素血症中的作用及其调控机制

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摘要 对高胰岛素血症的特征,胰岛β细胞瘦素抵抗在高胰岛素血症发生发展中的关键作用,以及通过SOCS3基因过表达的瘦素抵抗分子调控机制进行介绍,为寻找以胰岛β细胞JAK-STAT信号转导途径作为高胰岛素血症治疗的新分子靶点的特异药物提供基础。
出处 《中学生物教学》 北大核心 2022年第32期48-50,共3页 Teaching of Middle School Biology
基金 国家高技术研究发展863计划(2012AA021701) 江苏省高校重点建设实验室项目[苏教科(2016)8]
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