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腺苷抗心肌损伤及心肌肥厚的研究进展 被引量:1

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摘要 近5年来研究腺苷心肌保护作用的机制主要集中于受体,腺苷除了通过与其A1、A2A、A2B、A3受体结合,拮抗儿茶酚胺的心脏毒性、抑制白细胞及炎症因子的释放、抗氧化应激、诱导一氧化氮(nitricoxide,NO)生成、激活ATP敏感性钾通道、调节心肌肌浆网的钙转运、远隔保护等7个方面发挥抗心肌损伤作用外,还可以通过A1受体介导抗心肌肥厚效应。本文就腺苷抗心肌损伤及心肌肥厚的研究进展作一综述。
出处 《岭南心血管病杂志》 2006年第5期377-380,共4页 South China Journal of Cardiovascular Diseases
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参考文献28

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同被引文献12

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