1[1]Koonin AJ.The aetiology of keloids:a review of the literature and a new hypothesis[J].South Afr Med J,1964,38:913-918.
2[2]Marneros AG,Norris J E,Olsen B R,et al.Clinical genetics of familial keloids[J].Arch Dermatol,2001,137 (11):1429-1434.
3[3]David W,Friedman,Charles.et al.Regulation of collagen gene expression in keloids and hypirtro Phic scar[J].J Surg.Res,1993:55:214-222.
4[4]Sappino AP,Scburch W,Gabbiani G.Diferentitation rePertoire of fibroblastic cells:expression of cytoskcletal Proteins as marker of Phenotypic modulations[J].Lab Invest,1990:63:144.
5[5]Gross J.Getting to mammalian wound repair and amphibian limb regeneration:a mechan istic link in the early events[J].Wound Rep Reg,1996,4(2):190-202.
6[6]Lekic P C,Pender N,Mcculloch C A.Is fibroblast heterogeneity relevant to the heath,disease,and treatments of periodontal tissues[J].Crit Rev Oral Biol Med,1997,8(3):253-268.
8[8]Lin RY,Sullivan KM,Argenta PA,et al.Exogenous transforming growth factors-beta amplifies its own expression and induces scar formation in a model of human fetal skin repair[J].Ann surg,1995,222:146-154.
9[9]Joseph P,Grande J P.Role of transforming growth factor-β= in tissue injury and repair[J].Proc Soc Exp Biol Med,1997,214:27-40.
10[10]Yang L,Chai Y B,Li H Y,et al.Expression of Smad3 and Smad7mRNA of TGF-β1 in keloid-derived fibroblasts[J].Chin J Clin Reha-bil,2003,7(14):2004-2005.
