1[1]Sen R,Baltimore D.Multiple nuclear factors interact with the immunoglobulin enhancer sequences[J].Cell,1986,46 (5):705-716
2[2]Ghosh S,May M.J.and Kopp E.B.NF-kappa B and Rel proteins:evolutionarily conserved mediators of immune responses[J].Annu Rev Immunol.1998(16):225-260
3[3]Malek S,Chen Y,Huxford,T.and Ghosh,G.IkappaBbeta,but not IkappaBalpha,functions as a classical cytoplasmic inhibitor of NF-kappaB dimers by masking both NF-kappaB nuclear localization sequences in resting cells[J].J.Biol.Chem.2001(276):45225-4523
4[4]Yamamoto,Y.and Gaynor,R.B.IkappaBkinases:key regulators of the NF-kappaB pathway[J].Trends Biochem.Sci.2004 (29):72-79
5[5]Yamazaki S,Muta T,Takeshige K.A novel IκB protein,IκB-zeta,induced by proinflammatory stimuli,negatively regulates nuclear factor-κB in thenuclei[J].J Biol Chem,2001,276(29):27657-27662
6[6]Chen Lf,Fisce W,Verdin E,et al.Duration of nuclear NF-κB action regulated by reversible acetylation[J].Science,2001,293(5535):1653-1657
7[7]Brand K,Page S,Rogler G,Bartsch A,et al.Activated transcription factor nuclear factor-kappa B is present in the atherosclerotic lesion[J].J Clin Invest,1996,97(7):lT15-1722
9[9]Edwin Kanters,Marion J.J.Gijbels,Ingeborg van der Made,Hematopoietic NF-κB1 deficiency results in small atherosclerotic lesions with an inflammatory phenotypeC[J].Blood,1 February 2004,Vol.103,No.3,pp.934-940
