1[1]Majetschak M,Krehmeier U,Ostroverkh L,et al.Alterations in leukocyte function following surgical trauma:differentiation of distinct reaction types and association with tumor necrosis factor gene polymorphisms.Clin Diagn Lab Immunol,2005,12:296-303.
2[2]Feili-Hariri M,Falkner DH,Morel PA.Polarization of naive T cells into Th1 or Th2 by distinct cytokine-driven murine dendritic cell populations:implications for immunotherapy.J Leukoc Biol,2005,78:656-664.
3[3]Singh RA,Zhang JZ.Differential activation of ERK,p38,and JNK required for Th1 and Th2 deviation in myelin-reactive T cells induced by altered peptide ligand.J Immunol,2004,173:7299-7307.
4[4]Weighardt H,Heidecke CD,Westerholt A,et al.Impaired monocyte IL-12 production before surgery as a predictive factor for the lethal outcome of postoperative sepsis.Ann Surg,2002,235:560-567.
5[5]Matsukawa A,Kudoh S,Sano G,et al.Absence of CC chemokine receptor 8 enhances innate immunity during septic peritonitis.FASEB J,2006,20:302-324.
6[6]Watanabe H,Kubo M,Numata K,et al.Overexpression of suppressor of cytokine signaling-5 in T cells augments innate immunity during septic peritonitis.J Immunol,2006,177:8650-8657.
7[7]Faunce DE,Gamelli RL,Choudhry MA,et al.A role for CD1d-restricted NKT cells in injury-associated T cell suppression.J Leukoc Biol,2003,73:747-755.
8[8]Chen Z,Lund R,Aittokallio T,et al.Identification of novel IL-4/Stat6-regulated genes in T lymphocytes.J Immunol,2003,171:3627-3635.
9[9]Min B,Prout M,Hu-Li J,et al.Basophils produce IL-4 and accumulate in tissues after infection with a Th2-inducing parasite.J Exp Med,2004,200:507-517.
10[10]Nemeth ZH,Lutz CS,Csoka B,et al.Adenosine augments IL-10 production by macrophages through an A2B receptor-mediated posttranscriptional mechanism.J Immunol,2005,175:8260-8270.
