1Tuomi T,Groop LC,Zimmet PZ,et al.Autobodies to glutamic acid decarboxylase reveal latent autoimmune diabetes mellitus in adults with a non-insulindependent onset of disease[J].Diabetes,1993,42:3595.
2Tuomi T,Carlsson A,Li H,et al.Clinical and genetic characteristics of type 2 diabetes with and without GAD antibodies[J].Diabetes,1999,48:150-157.
3Pozzilli P,Mario UD.Autoimmune diabetes not requiring insulin at diagnosis (latent autoimmune diabetes of the adult)[J].Diabetes Care,2001,24:1460-1467.
4Fukui M,Nakamura N,Nakano K,et al.Cellular immune response to GAD in type 1 diabetes with residualβ-cell function[J].Diabetes Care,1999,22:1758.
8Williams AJ,Bingley PJ,Moore WP,et al.Islet autoantibodies,nationalityand gender:a multinational screening study in first-degree relatives of pa-tients with Type I diabetes[J].Diabetologia,2002;45(2):217-23.
9Achenbach P,Warncke K,Reiter J,et al.Stratification of type 1 diabetesrisk on the basis of islet autoantibody characteristics[J].Diabetes,2004;53(2):384-92.
10Li X,Zhou ZG,Huang G,et al.Optimal cutoff point of glutamate decar-boxylase antibody titers in differentiating two subtypes of adult-onset la-tent autoimmune diabetes[J].Ann N Y Acad Sci,2004;1037:122-6.
