1[1]Kennedy SH, Evans KR, Kruger S, et al. Changes in regional brain glucose metabolism measured with positron emission tomography after paroxetine treatment of major depression. Am J Psychiatry, 2001, 158(6) :899~905
2[2]Ketter TA, George MS, Kimbrell TA, et al. Functional brain imaging, limbic function and affective disorers. Neuroscientist, 1996,2:55 ~ 65
3[3]Kimbrell TA, Ketter TA, George MS, et al. Regional cerebral glucose utilization in patients with a range of severities of unipolar depression.Biol Psychiatry, 2002, 51 (3) : 237 ~ 252
4[4]Videbech P. PET measurements of brain glucose metabolism and blood flow in major depressive disorder. Acta Psychiatry Scand,2000, 101(1) : 11 ~20
5[5]Brody AL, Saxena S, Silverman DHS, et al. Brain metabolic changes in major depressive disorder from pre to post-treatment with paroxetine. Psychiatry Res Neuroimaging, 1999, 1 : 127 ~ 139
7[7]Mayberg HS, Branna SK, Tekell JL, et al. Regional metabolic effects of fluoxetine in major depression: serial changes and relationship to clinical reseponse. Biol Psychiatry, 2000, 48: 830 ~ 843
8[8]Drevets WC, Frank E, Price JC, et al. PET imaging of serotonin 1A recepoor binding in depresion. Biol Psychiatry, 1999, 46 (10) :1375 ~ 1387
9[9]Yatham LN, Liddle PF, Shiah IS, et al. Brain serotonin 2 receptors in major depression: a positron emission tomography study. Arch Gen Psychiatry, 2000, 57(9) : 850 ~858
10[10]Bremner JD, Ianis RM, Salomon RM, et al. Position emission tomography measurement of cerbral metabolic correlates of tryptophan deletion-induced depressive relapse. Arch Gen Psychiatry, 1997,54 (4):364 ~ 374
2CHEN L M, ZUO C T, HUANG Z M. Establishment of a Normal Brain 18F-FDG PET/CT Database [ J ]. clinical im- aging technology ,2010,25 ( 1 ) : 123 - 126.
3KIMBRELL T A, KETrER T A, GEORGE M S, et al. Regional cerebral glucore utilization in patients with a range of severities of unipolar depression [ J ]. Biol Psychi- atry, 2002,51 (3) :237 -252.