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心力衰竭的内质网应激机制 被引量:4

Mechanism of endoplasmic reticulum stress in heart failure
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摘要 心力衰竭是心血管疾病晚期共有的终末器官损害,疗效差,死亡率高,是临床上亟待解决的重大问题。长期、慢性神经内分泌细胞因子系统的激活引起的心肌重塑是心力衰竭发生的基本机制,防止和延缓心肌重塑的发展已经成为目前心力衰竭治疗的根本策略[1]。
作者 刘秀华
出处 《中国医学前沿杂志(电子版)》 2010年第1期6-12,共7页 Chinese Journal of the Frontiers of Medical Science(Electronic Version)
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参考文献37

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  • 2刘秀华,唐朝枢.重视心血管疾病中的内质网应激机制——从细胞应激的基本反应到临床防治的思考[J].中华医学杂志,2009,89(32):2233-2234. 被引量:4
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二级参考文献93

共引文献89

同被引文献58

  • 1刘唐威,王秋雁,伍伟锋.内质网伴侣蛋白在小鼠病毒性心肌炎心肌细胞的表达及其与细胞凋亡的关系[J].广西医科大学学报,2005,22(2):157-159. 被引量:9
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  • 3刘玉清,李佳,张禅那,宋晓东,刘晓雷,惠汝太.病毒性心肌炎细胞感染模型的建立及实验研究[J].中国分子心脏病学杂志,2002,2(4):25-29. 被引量:11
  • 4关丽英,许彩民,潘华珍.内质网应激介导的细胞凋亡[J].生物化学与生物物理进展,2007,34(11):1136-1141. 被引量:74
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