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血管内皮素、血清纤维蛋白原与脑梗死的相关性 被引量:13

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摘要 血管内皮素(ET)升高可引起缺血区侧支血管强烈持久收缩、痉挛,加重梗死区缺血并刺激大量兴奋氨基酸及炎性因子的释放促使脑梗死细胞坏死呈级联反应,纤维蛋白原(FBI)参与凝血过程,同时也是一种应激蛋白,参与动脉粥样硬化的进程。本研究对急性脑梗死患者血浆ET及血清FIB水平进行对照研究,从分子生物学水平寻求治疗急性脑梗死的理论依据。
出处 《中国老年学杂志》 CAS CSCD 北大核心 2014年第4期1069-1070,共2页 Chinese Journal of Gerontology
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