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Emodin suppresses LPS-induced proinflammatory responses and nuclear factor-B activation by disruption of lipid rafts and TLR-4 recruitment in endothelial cells

Emodin suppresses LPS-induced proinflammatory responses and nuclear factor-B activation by disruption of lipid rafts and TLR-4 recruitment in endothelial cells
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摘要 Emodin [1,3,8-Trihydroxy-6-methylanthraquinone] has been reported to exhibit vascular anti-inflammatory properties.However,the relevant anti-inflammatory mechanisms are not well understood.The present study was design to explore the molecular target(s) of emodin
出处 《医用生物力学》 EI CAS CSCD 2009年第S1期122-122,共1页 Journal of Medical Biomechanics
基金 supported by National Natural Science Foundation of China,30700151 Youth Investigator Fund from UESTC,Y02018023601062
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