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慢性肾衰继发性甲旁亢的病因及治疗研究新进展 被引量:3

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作者 湛冯岚
出处 《国际泌尿系统杂志》 2001年第S1期54-57,共4页 International Journal of Urology and Nephrology
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参考文献10

  • 1Brown EM,Gamba G,Riccardi D,et al. Nature . 1993
  • 2Coburn JW,Elangovan L,Goodman WG,et al. Kidney International . 1999
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  • 6Chang JM,Hwang SJ,Tsai JC,et al. Nephrology Dialysis Transplantation . 1999
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同被引文献15

  • 1<活性维生素D的合理应用>专家协作组.活性维生素D在慢性肾脏病继发性甲旁亢中合理应用的专家共识(修订版)[J].中华肾脏病杂志,2005,21(11):698-699. 被引量:97
  • 2欧三桃,赵安菊,黄颂敏.继发性甲旁亢对慢性肾衰竭患者的影响及治疗进展[J].国际泌尿系统杂志,2006,26(6):831-834. 被引量:9
  • 3Ruiz OM,Largo R,Bustos C et al.Platelet-activating factor stimulates gene expression and synthesis of matrix proteins in cultured rat and human mesangial cells: Role of TGF-beta[].Journal of the American Society of Nephrology.1997
  • 4Short AD,Taylor CW.Parathyroid hormone controls the size of the intra-cellular Ca2+ stores available to receptors linked to inositol trisphosphate formation[].Journal of Biological Chemistry.2000
  • 5Wu Y,Kumar R.Parathyroid hormone regulates transforming growth factor betal and beta2 synthesis in osteoblasts via divergent signaling pathways[].Journal of Bone and Mineral Research.2000
  • 6StuderRK,Craven PA,DeRubertis FR.Antioxidant inhibition of protein ki-nase C-signaled increases in transforming growth factor-beta in mesangial cells[].Metabolism.1997
  • 7Pawluczyk IZ,Harris KP.Cholesterol feeding activates macrophages to up-regulate rat mesangial cell fibronectin production[].Nephrology Dialysis Transplantation.2000
  • 8R iser BL,DenichiloM,Cortes P,et al.Regulation of connective tissue growth factor activity in cultured ratmesangial cells and its expression in experimental diabetic glomerulosclerosis[].Journal of the American Society of Nephrology.2000
  • 9Kim SI,Han DC,Lee HB.Lovastatin inhibits transforming growth factor-betal expression in diabetic rat glomeruli and cultured rat mesangial cells[].Journal of the American Society of Nephrology.2000
  • 10Sugiura T,Imai E,Moriyama T et al.Calcium channel blockers inhibit proliferation and matrix production in rat mesangial cells Possible mechanism of suppression of AP-1 and CREB activities[].Nephron.2000

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