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髓系白血病原代细胞和肿瘤细胞株ATM转录水平的研究 被引量:5

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出处 《中华血液学杂志》 CAS CSCD 北大核心 2004年第6期379-380,共2页 Chinese Journal of Hematology
基金 国家自然科学基金资助项目 ( 3 980 0 14 9)
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参考文献7

  • 1Stoppa-Lyonnet D, Soulier J,Lauge A,et al. Inactivation of the ATM gene in T-cell prolymphocytic leukemias.Blood, 1998,91:3920-3926.
  • 2Starostik P,Manshouri T,O'Brien S,et al. Deficiency of the ATM protein expression defines an aggressive subgroup of B-cell chronic lymphocytic leukemia. Cancer Res, 1998,58:4552-4557.
  • 3Schaffner C,Stilgenbauer S,Rappold GA,et al. Somatic ATM mutations indicate a pathogenic role of ATM in B-cell chronic lymphocytic leukemia.Blood,1999,94:748-753.
  • 4Stankovic T,Weber P,Stewart G,et al. Inactivation of ataxia telangiectasia mutated gene in B-cell chronic lymphocytic leukaemia.Lancet,1999,353:26-29.
  • 5Haidar MA,Kantarjian H,Manshouri T,et al. ATM gene deletion in patients with adult acute lymphoblastic leukemia.Cancer,2000,88:1057-1062.
  • 6Wen-Sheng W. ERK signaling pathway is involved in p15INK4b/p16INK4a expression and HepG2 growth inhibition triggered by TPA and Saikosaponin a.Oncogene,2003, 22: 955-963.
  • 7Melo JV,Kumberova A,van Dijk AG,et al.Investigation on the role of the ATM gene in chronic myeloid leukaemia.Leukemia,2001,15:1448-1450.

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