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肝硬化患者血清三项参数的变化及其意义 被引量:1

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出处 《中国医师杂志》 CAS 2004年第9期1258-1259,共2页 Journal of Chinese Physician
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  • 1中华医学会传染病与,寄生虫病学分会,肝病学分会.病毒性肝炎防治方案[J].中华肝脏病杂志,2000,8(6):324-329. 被引量:14010
  • 2Fujimoto M, Uemura M, Nakatani Y, et al. Plasma endotoxin and serum cytokine levels in patients with alcoholic hepatitis:relation to severity of liver disturbance[J]. Alcohol Clin Exp Res, 2000, 24(4 Suppl): 48S-54S
  • 3Ariki N, Morimoto Y, Yagi T, et al. Activated T cells and soluble molecules in the portal venous blood of patients with cholestatic and hepatitis C virus-positive liver cirrhosis. Possible promotion of Fas/FasL-mediated apoptosis in the bile-duct cells an
  • 4Vaillant B, Chiaramonte MG, Cheever AW, et al. Regulation of hepatic fibrosis and extracellular matrix genes by the th response: new insight into the role of tissue inhibitors of matrix metalloproteinases[J]. J Immunol, 2001, 167(12):7017-7026
  • 5Bahr MJ, el Menuawy M, Boeker KH, et al. Cytokine gene polymorphisms and the susceptibility to liver cirrhosis in patients with chronic hepatitis C[J]. Liver Int,2003, 23(6):420-425
  • 6Shawcross DL, Davies NA, Williams R, et al. Systemic inflammatory response exacerbates the neuropsychological effects of induced hyperammonemia in cirrhosis[J]. J Hepatol,2004, 40(2): 247-254
  • 7Tsutsui H, Adachi K, Seki E, et al. Cytokine-induced inflammatory liver injuries[J]. Curr Mol Med,2003, 3(6): 545-559

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