1Jaffrey SR, Erdjument-Bromage H, Ferris CD, et al. Protein S-nitrosylation: a physiological signal for neuronal nitric oxide. Nat Cell Biol,2001,3:193-197.
2Tunter T. Signaling-2000 and beyond. Cell,2000,100:113-127.
3Qye I. Ketamine analgesia, NMDA receptors and the gates of perception.Acta Anaesthesiol Scand, 1998,42:747-749.
4Miyawaki I, Nakamura K, Yokubol B, et al. Suppression of cyclic guanosine monophosphate formation in rat cerebellar slices by propofol,ketamine and midazolam. Can J Anaesth, 1997,44:1301-1307.
5Galley HF, Webster NR. Brain nitric oxide synthase activity is decreased by intravenous anesthetics. Anesth Analg, 1996,83:591-594.
6Li CY, Chou TC, Wong CS, et al. Ketamine inhibits nitric oxide synthase in lipopolysaccharide-treated rat alveolar macrophages. Can J Anaesth, 1997,44(9) :989-995.
7Tonner PH, Scholz J, Lamberz L, et al. Inhibition of nitric oxide synthase decreases anesthetic requirements of intravenous anesthetics in Xenopus laevis. Anesthesiology, 1997,87:1479-1485.
8Wu J, Kikuchi T, Wang Y, et al. NOx- concentrations in the rat hippocampus and striatum have no direct relationship to anaesthesia induced by ketamine. Br J Anaesth,2000,84:183-189.
9Bulutcu F, Dogrul A, Guc MO. The involvement of nitric oxide in the analgesic effects of ketamine. Life Science,2002,71:841-853.
10Miyawaki I, Nakamura K, Terasako K, et al. Modification of endothelium-dependent relaxation by propofol, ketamine, and midazolam. Anesth Analg, 1995,81:474-479.
