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老年钙化性主动脉瓣病变的临床特点和外科治疗 被引量:3

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出处 《中华心血管病杂志》 CAS CSCD 北大核心 2004年第5期434-435,共2页 Chinese Journal of Cardiology
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  • 1Otto C M. Calcific aortic stenosis time to look more closely at the valve[J]. N Engl J Med,2008,359:1395- 1398.
  • 2Freeman R V, Otto C M. Spectrum of caleific aortic valve disease: pathogenesis, disease progression, and treatment strafe gies[J]. Circulation,2005,111:3316 -3326.
  • 3El Kahi C G,Atiyeh B S,Abdallah Hajj Hussein I,Jurjus R,Dibo S A,Jurjus A,et al. Modulation of wound contracture alphasmooth muscle actin and multispecific vitronectin receptor integrinalphavbeta3 in the rabbit's experimental model [J]. Int Wound J, 2009,6 : 214-224.
  • 4Baoren Z. Heart valve surgery in China: yesterday and today [J]. Heart Lung Circ, 2001,10:A11-A16.
  • 5Nigam V, Srivastava D. Notchl represses osteogenie pathways in aortic valve cells[J]. J Mol Cell Cardiol, 2009, 47:828 -834.
  • 6Yang X,Meng X,Su X,Mauchley D C,Ao L,Cleveland J C Jr, et al. Bone morphogenie protein 2 induces Runx2 and osteopontin expression in human aortic valve interstitial cells: role of Smadl and extracellular signal regulated kinase 1/2[J]. J Thorac Cardiovasc Surg, 2009,138 : 1008-1015.
  • 7Gu X, Masters K S. Role of the MAPK/ERK pathway in valvular interstitial cell calcification[J]. Am J Physiol Heart Cire Physiol,2009,296 : H1748- H1757.
  • 8Yip C Y, Chen J H, Zhao R, Simmons C; A. Calcification by valve interstitial cells is regulated by the stiffness of the extracellular matrix [J]. Arterioscler Thromb Vasc Biol, 2009,29:936-942.
  • 9Xu Y,Pritzker K P, Cruz T F. Characterization of ehondrocyte alkaline phosphatase as a potential mediator in the dissolution of calcium pyrophosphate dihydrate crystals[J]. J Rheumatol, 1994,21:912- 919.
  • 10Han L, Gotlieb A I.Fibroblast growth factor-2 promotes in vitro heart valve interstitial cell repair through the Akt1 pathway[J].Cardiovasc Pathol, 2012,21: 382-389.

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