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Smad2/3和Smad4在前列腺癌组织中的表达 被引量:5

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出处 《临床泌尿外科杂志》 2004年第10期621-622,共2页 Journal of Clinical Urology
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  • 1Caestecker M P, Piek E, Roberts A. Role of growth factor-βsignaling in cancer. J Natl Cancer Inst, 2000,92: 1388-1402.
  • 2Wilentz R E, Su G H, Dai J L, et al. Immunohistochemical labeling for DPC4 mirrors genetic status in pancreatic adenocarcinomas: a new marker of DPC4 inactivation. Am J Pathol, 2000, 156: 37-43.
  • 3Maliekal T T, Antony M L, Nair A, et al. Loss of expression, and mutations of Smad 2 and Smad 4 in human cervical cancer. Oncogene,2003, 22: 4889- 4897.
  • 4Piestrzeniewicz-Ulanska D, Brys M, Semczuk A, et al.Expression and intracellular localization of Smad proteins in human endometrial cancer. Oncol Rep, 2003,10: 1539-1544.
  • 5Nicolas F J, Hill C S. Attenuation of the TGF-betaSmad signaling pathway in pancreatic tumor cells confers resistance to TGF-beta-induced growth arrest. Oncogene, 2003, 22: 3698-3711.
  • 6van der Poel H G, Hanrahan C, Zhong H, et al. Rapamycin induces Smad activity in prostate cancer cell lines. Urol Res, 2003, 30: 380-386.
  • 7Hayes S A, Huang X, Kambhampati S, et al. p38 MAP kinase modulates Smad-dependent changes in human prostate cell adhesion. Oncogene, 2003, 22:4841-4850.

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