1[1]Farrar MA,Schreiber RD.The molecular cell biology of interferon-γ and its receptor.Annu Rev Immunol,1993,11:571-611.
2[2]Pellegrini S,Schindler C.Early events in signaling by interferons.Trends Biochem Sci,1993,18:338-342.
3[3]Hunang S,Hendriks W,Althage A,et al.Immune response in mice that lack the interferon-γ receptor.Science,1993,259:1742-1745.
4[4]Darnell JE Jr,Kerr IM,Stark GR.Jak-STAT pathways and transcriptional activation in response to IFNs and other extracellular signaling proteins.Sicience,1994,264:1415-1421.
5[5]Ihle JN.STATs:signal transducers and activators of transcription.Cell,1996,84:331-334.
6[6]Schindler C,Damell JE Jr.Transcriptional responses to polypeptide ligands:the JAK-STAT pathway.Annu Rev Biochem,1995,64:621-651.
7[7]Liew FY,Li Y,Severn A,et al.A possible novel pathway of regulation by murine T helper type-2(Th2)cells of a Th1 cell activity via the modulation of the induction of nitric oxide synthase on macrophages.Eur J Immunol,1991,21:2489-2494.
8[8]Singer DS,Maguire JE.Regulation of the expression of class I MHC genes.Crit Rev Immunol,1990,10:235-257.
9[9]Miyamoto M,Fujita T,Kimura Y,et al.Regulated expression of a gene encoding anuclear factor,IRF-1,that specifically binds to IFN-β gene regulatory elements.Cell,1988,54:903-913.
10[10]Pine R,Decker T,Kessler DS,et al.Purification and cloning of interferon-stimulated gene factor 2(ISGF2):ISGF2(IRF-1) can bind to the promoters of both β-interferon and interferon-stimulated genes but is not a primary transcriptional activator of either.Mol cell Biol,1990,10:2448-2457.
