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转化生长因子β与肝纤维化的关系 被引量:8

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作者 陈霞 李昌平
出处 《临床肝胆病杂志》 CAS 北大核心 2004年第6期379-381,共3页 Journal of Clinical Hepatology
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  • 1Kinnman N, Andersson V, Hultcrantz R, et al, In situ expression of fransforming growth factor- beta 1 - 3 latent transforming growth factorbata binding protein and tumor necrosis factor- alpha in liver tissue from patients with chronic hepatitis C[ J]. Sc
  • 2Mamura M, Inagaki Y. Constitutive phosphorylation and Nuclear localization of Smad3 are correlated with increased collagen gene transcription in activated hepatic stellates cells[J] .J Cell Phy, 2001, 187:117 - 123.
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  • 4Flisiak R, Maxwell P. Plasma tissue inhibitior of metalloproteinases- 1 and transforming growth fatctor beta - 1-Possible non - invasive biomakers of hepatic fibrosis in patients with chronic B and C hepatitic[J] .Hpatoenterology, 2002,49(4): 1369 - 1372.
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  • 6Sylke, Roth-Eichhorn, Kuhl K, et al. Subcellular localization of (latent) transforming growth factor β and the latent TGF- β and the latent TGF- β binding protein in rat hepatocytes and hepatic stellate cells[ J].Hepatology, 1998,28(6): 1588 - 1596.
  • 7Liu C, Gala ID, Suenson ES. Smad2 and 3 are differentially activated by transforming growth factor- Beta(TGF-beta) in quiescent and activated hepatic stellate cells[J] .J Biol Chem, 2003,278(13) :11721 - 8.
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