1Urunuela A, Sevillano S, de la Mano AM, et al. Time-course of oxygen free radical production in acinar ceils duringacute pancreatitis induced by pancreatic duct obstruction[J]. Biochim Biophys Acta,2002, 1588(2) : 159-164.
2Demols A, Van Laethem JL, Quertinmont E, et al. N-Acetylcysteine decreases severity of acute pancreatitis in mice [J]. Pancreas, 2000, 20(2) : 161-169.
3Hyeyoung K, Jeong YS, Kwan HR, et al. Suppression of NF-kB activationg and cytokine prodution by N-Acetylcysteine in pancreatic acinar cells[J]. Free Radical Biol Med,2000,29(9):674-683.
4Vaquero E, Gukovsky I, Zaninovic V, et al. Locahzed pancreatic NF-kappaB activation and inflammatory response in taurocholate-induced pancreatitis[J] . Am J Physiol-Gastrointest Liver Physiol, 2001,280(6) :G1197-1208.
5Gukovsky I, Gukovskaya AS, Blinman TA, et al. Early NF-kappaB activation is associated with hormone-induced pancreatifis[ J]. Am J Physiol,1998,275(6 Pt 1):1402-1414.
6Sevillano S, De la Mano AM, De Dios I, et al. Major pathological mechanisms of acute pancreatifis are prevented by N-Acetylcysteine[J]. Digestion, 2003,68 ( 1 ) : 34-40.
7Sevillano S, De la Mano AM, Manso MA, et al. N-acetylcysteine prevents intra-acinar oxygen free radical production in pancreatic duct obstructlon-induced acute pancreatitls [ J] . Biochim Biophys Acta, 2003,1639(3) : 177-184.
8Sevillano S, De Dios I, De la Mano AM, et al. N-Acetylcysteine induces beneficial changes in the acinar cell cycle progression in the course of acute pancreatitis [ J ]. Cell Proliferation, 2003,36 (5) : 279-289.
9Jun Y, Murphy C, Woody D, et al. Evidence of a central role for p38 map kinase induction of tumor necrosis factor alpha in pancreafitis-associated pulmonary injury[J]. Surgery, 1999,126(2) :216-222.
10Woody D,Jun Y, Haichao W, et cd. Inhibition of p38 mitogen activate kinase attenuates the severity of pancreatifis-induced adult respiratory distress syndrome[J]. Crit Care Med,2000,28(7):2567-2572.
