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挥发性麻醉剂预处理对器官缺血再灌注损伤保护作用的研究 被引量:4

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出处 《医学综述》 2005年第1期78-80,共3页 Medical Recapitulate
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  • 1Kapinya KJ, [,owl D, Futterer C, et o2.Tolerance against ischemic neuronal injury can be induced by volatile anesthetics and is inducible NO synthase dependent[ J]. Stroke ,2002,33(7) : 1889-1898.
  • 2Pifiou V, Chiari P, Lhuillier F, et al. Pharmacological preconditioning :comparison of desflurane, sevoflurane, isoflurane and halothane in rabbit myocardium [ J ]. Br J Anaesth ,2002,89 (3) : 486-491.
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  • 4Kehl F, Krolikowski JG, Weihrauch D, et al. N-acetylcysteine restores isoflurane-induced preconditioning against myocardial infarction during hyperglycemia [ J ]. Anesthesiology, 2003,98 ( 6 ) : 1384-1390.
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  • 6Chen Q, Camara AK, An J, et al. Sevoflurane preconditioning before moderate hypothermic ischemia protects against cytosolic [ Ca^2 + ] loading and myocardial damage in part via mitochondrial KAte channels[J].Anesthesiology, 2002,97 ( 4 ) : 912-920.
  • 7Kevin LG, Novalija E, Riess ML, et al. Sevoflurane exposure generates superoxide but leads to decreased superoxide during ischemia and reperfusion in isolated hearts [ J]. Anesth Analg, 2003,96 ( 4 ) : 949-955.
  • 8Novalija E, Kevin LG, Eells JT, et al. Anesthetic preconditioning improves adenosine triphosphate synthesis and reduces reactive oxygen species formation in mitochondria after ischemia by a redox dependent mechanism[J]. Anesthesiology,2003,98(5) :1155-1163.
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  • 10Kwok WM, Martineni AT, Fujimoto K, et al. Differential modulation of the cardiac adenosine triphosphate-sensitive potassium channel by isoflurane and halothane[ J]. Anesthesiology ,2002,97( 1 ) :50-56.

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