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基质金属蛋白酶和炎症性肠病 被引量:1

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出处 《临床消化病杂志》 2005年第1期47-48,共2页 Chinese Journal of Clinical Gastroenterology
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同被引文献10

  • 1Morris GP, Beck PL, Herridge MS, et al. Hapten-induced model of chronic inflammation and ulceration in the rat colon. Gastroenterology, 1989,96:795-803.
  • 2Medina C, Videla S, Radomski A, et al. Therapeutic effect of phenanthroline in two rat models of inflammatory bowel disease. Scand J Gastroenterol, 2001,36:1314-1319.
  • 3Park JM, Adam RM, Peters CA, et al. AP-1 mediates stretch-induced expression of HB-EGF in bladder smooth muscle cells. Am J Physiol, 1999, 277 :C294-301.
  • 4Yan M, Schneider J, Gear R, et al. Expression of angiogenic factors is upregulated in DMBA-induced rat mammary pathologies. Pathobiology, 2004, 71:253-260.
  • 5Deschner J, Rath-Deschner B, Agarwal S. Regulation of matrix metalloprot-einase expression by dynamic tensile strain in rat fibrochondrocytes. Osteoart-hritis Cartilage, 2006, 14:264-272.
  • 6Suzuki A, Hanada T, Mitsuyama K, et al. CIS3/SOCS3/SSI3 plays a negativeregulatory role in STAT3 activation and intestinal inflammation. J Exp Med, 2001, 193:471-481.
  • 7Lovato P, Brender C, Agnholt J, et al. Constitutive STAT3 Activation in intestinal T Cells from Patients with Crohn's Disease. J Biol Chem, 2003,278 : 16777-16781.
  • 8Alessandro M, Patrizia D, Alessandra C, et al. Signal transducers and activators of Transcription 3 signaling pathway an essential mediator of Inflammatory bowel disease and other forms of intestinal inflammation. Inflamm Bowel Dis, 2005,11:91-98.
  • 9Yamamoto M, Yoshizaki K, Kishimoto T, et al. IL-6 is required for the development of Th1 cell-mediated murine colitis. J Immunol, 2000, 164:4878-4882
  • 10姚胜,姚咏明,李红云,董宁,于燕,梁华平.抑制JAK/STAT通路对烫伤后金黄色葡萄球菌脓毒症大鼠肝损害的影响[J].中国危重病急救医学,2002,14(6):336-339. 被引量:15

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