2Kestila M, Lenkkeri U, Manikko M, et al. Positionally cloned gene for a novel glomerular protein - nephrin is mutated in congenital nephrotie syndrome. Mol Cell, 1998, 1 (4) : 575 -582.
3Bertelli R, Ginevri F, Caridi G, et al. Recurrence of focal segmental glomerulosclerosis after renal transplantation in patients with mutations of podocin. Am J Kidney Dis,2003,41(6) : 1314 - 1321.
4Schwarz K, Simons M, Reiser J, et al. Podocin, a raft - associated component of the glomerular slit diaphragm, interacts with CD2AP and nephrin. J Clin Invest, 2001,108 ( 11 ) :1621 - 1629.
5Shih NY, Li J, Cotran R, et al. CD2AP localizes to the slit diaphragm and binds to nephrin via a novel C - terminal domain. Am J Pathol, 2001,159(6) :2303 - 2308.
6Dustin ML, Olszowy MW, Holdorf AD, et al. A novel adaptor protein orchestrates receptor patterning and cytoskeletal polarity in T - cell contacts. Cell, 1998,94(5) :667 - 677.
7Kim JM, Wu H, Green G, et al. CD2-associated protein haploinsufficiency is linked to glomerular disease susceptibility. Science,2003,300(5623) : 1298 - 1300.
8Sellin L, Huber TB, Gerke P, et al. NEPH1 defines a novel family of podocin interacting proteins. FASEB J, 2003, 17(1):115- 117.
9Schnabel E,Anderson JM, Farquhar MG. The tight junction protein ZO - 1 is concentrated along slit diaphragms of the glomerular epithelium. J Cell Biol, 1990, 111 ( 3 ) : 1255 -1263.
10Inoue T, Yaoita E, Kurihara H, et al. FAT is a component of glomemlar slit diaphragms. Kidney hat ,2001,59(3) :1003 -1012.
二级参考文献24
1Hiromura K, Haseley LA, Zhang P,et al.Podocyte expression of the CDK-inhibitor p57 during development and disease. Kidney Int,2001,60(6):2235-2246.
2Petermann AT, Pippin J, Hiromura K,et al.Mitotic cell cycle proteins increase in podocytes despite lack of proliferation. Kidney Int,2003,63(1):113-122.
3Huh W, Kim DJ, Kim MK,et al.Expression of nephrin in acquired human glomerular disease. Nephrol Dial Transplant,2002,17(3):478-484.
4Doublier S, Ruotsalainen V, Salvidio G,et al.Nephrin redistribution on podocytes is a potential mechanism for proteinuria in patients with primary acquired nephrotic syndrome. Am J Pathol,2001,158(5):1723-1731.
6Saran AM, Yuan H, Takeuchi E,et al.Complement mediates nephrin redistribution and actin dissociation in experimental membranous nephropathy. Kidney Int,2003,64(6):2072-2078.
7Yuan H, Takeuchi E, Taylor GA,et al. Nephrin dissociates from actin, and its expression is reduced in early experimental membranous nephropathy. J Am Soc Nephrol,2002,13(4):946-956.
8Yang Y, Gubler MC, Beaufils H. Dysregulation of podocyte phenotype in idiopathic collapsing glomerulopathy and HIV-associated nephropathy. Nephron,2002,91(3):416-423.
9Hara M, Yanagihara T, Kihara I. Urinary podocytes in primary focal segmental glomerulosclerosis.Nephron,2001,89(3):342-347.
10Pavenstadt H,Kriz W,Kretzler M.Cell biology of the glomerular podocyte .The American physiological society,2003,83:253-307.
5Chen S,Lee JS,Iglesias-de la Cruz MC,et al.Angiotemin Ⅱ stimulares alpha 3(Ⅳ)collagen production in mouse podocytes via TGFbeta and VECF signalling:implications for diabetic glomerulopathy.Nephrol Dial Transolant,2005,20(7):1320.
6Iglesas-de la Cruz MC,Ziyadeh FN.Effects of high glucose and TGF-betal on the expression of colleen Ⅳ and vascular endothelial growthy factor in mouse poclocyte Kidney Int,2002,62(3):901.
7Timpl R Structure and biological activity of bascment membrance proteins.Eur J Biochem,1989,180(3):487.
8Feehally J,Allen AC.Pathogenesis of IgA-nephropathy.An Med Interne Paris,1999,150(2):91.
9Ralf Westerhuis,Masashi Kitahara,Jrgen Floege.IgA-Neohropathy:how doe8 IgA activate the mesangial cell?Clin Exp Nephrol,2001.95(5):1.
