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冠心病患者红细胞变形能力白细胞活性及黏附性的变化 被引量:1

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作者 刘登海
机构地区 枣庄市血站
出处 《中国基层医药》 CAS 2005年第1期104-105,共2页 Chinese Journal of Primary Medicine and Pharmacy
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  • 1谢景荣,齐文成,李俊英,李敬兰,曹金霓.常见血栓性疾病红细胞变形能力研究[J].天津医药,1995,23(11):663-665. 被引量:1
  • 2金永娟,朱文云.高脂血症循环白细胞自发活化作用的研究[J].中国微循环,1997,1(1X):29-32. 被引量:16
  • 3Buckley CD, Rainper GE, Bradfield PF, et al. Cell adhesion: more than just glus(review). Mol Membr Biol, 1998,15:167-176.
  • 4Hayashi J, lshida N, Sato H, et al. Effect of beraprost, a stable prostacyclin analogue,on red blood cell deformability impairment in the presence of hypercholesterolemia in rabbits. J Cardiovasc Pharmacol, 1996: 527-531.
  • 5Martinez M,Vaya A, Marti R,et al. Effect of HMG-CoA reductase inhibitors on red blood cell membrane lipids and haemorheological paiameters, in patients affected by familial hypercholesterolemia. Haaemostasis, 1996,26(suppl 4): 171-176.
  • 6Wolfgang Volker, Borszewski A, Unruh V, et al. Copperinduced inflammatory of rat corotid arteries mimic restenosis arterosclerosis-like neointima formation. Atherosclerosis, 1997,130:29.
  • 7Oguchi S, Dimayuga P, Zhu J, et al. Monoclonal antibody against vascular cell adhesion molecule-1 inhibits neointimal formtion after periadventitial carotid artery injury in genetically hypercholesterolemic mice. Arterioscler Thomb Vasc Biol, 2000,20:1729-1736.
  • 8Mehta J L, Li DY. Inflammation in ischemic heart disease:reponse to tissue injury or a pathogenetic villain?. Cardiovasc Res, 1999,43:291-299.

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