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医疗废物处置过程中相关问题的探讨 被引量:1

Discussion on the Development of Medical Refuse Disposal
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摘要 医院废物的处置工作是保证人民身体健康和环境的重要因素,同时也是经济可持续发展战略的重要保证。为此,针对我国2003年遭受突如其来的非典型肺炎的袭击,对医院废物处置工作中的一系列问题进行分析研究。现阶段处置医疗废物的有效措施是:积极推行无害化废物焚烧,防止二次污染;做好新形势下医疗废物管理工作,推动医疗废物焚烧健康发展;合理利用余热以及医疗废物的资源化。 This paper aimed at problems in hospital waste disposal after the SARS arised suddenly last year. We should sufficiently realize hospital waste treatment is a critical task now and in the future. Hospital waste treatment is important for protecting people’s health and our environment and promoting economy sustainable development. Now 1:we should encourage incineration without pollution and take efficiently measure to avoid pollution again 2: now improve management 3: utilize energy rationally and recycle the waste.
出处 《农机化研究》 北大核心 2005年第2期51-53,共3页 Journal of Agricultural Mechanization Research
关键词 医疗废物 焚烧 医院废物 资源化 无害化 二次污染 处置 经济可持续发展 重要因素 战略 hygieno engineering Science hospital waste summary non-pollution incineration recycles
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  • 1Robertson RW, Zhang L, Pasco DS, et al. Aryl hydrocarboninduced interaction at multiple DNA elements of diverse sequence-a multicomponent mechanism for activation of cytochrome P4501A1(CYP1A1)gene transfection. Nuleic. Acids. Res. 1994, 22(9): 1741-1749.
  • 2Enan E. and Masumura F. Identification of c-Src as integral componment of the cytosolic Ah receptor complex, transducing the signal of 2, 3, 7, 8-Tetrachlorodibenzo-p-dioxin (TCDD) through the protein phophorylation parthway. Biochem. Pharmacol. 1995, 52 (10): 1599-1612.
  • 3Blankenship A., and Matsumura F. 2, 3, 7, 8-Tetra-chlorodibenzo-p-dinxin-induced activation of a protein tyrosine kinase, pp60src, in murine hepatic cytosol using a cell-free system. Mol. Pharmacol. 1997, 52 (4): 667-675.
  • 4Kohle C, Gschaidmeier H, and Lauth D, et al. 2,3,7,8-Tetra-chlorodibenzo-p-dioxin(TCDD)-mediated membrane translocation of c-Src protein kinase in liver WB-F344 cells. Arch Toxicol. 1999. 73(3): 152-158
  • 5Enan E, Moran F, Vande Voort CA, et al. Mechanism of toxic action of 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) in cultured human luteinized granulosa cells. Reprod. Toxicol. 1996, 10(6): 497-508.
  • 6Hugh Olsen, Essan Enan, and Fumio Matsumura. Regulation of glucose transport in the NIN 3T3 L1 preadipocyte cell line by TCDD. Environmental Health Perspective. 1994, 102 (5): 454-468.
  • 7Seefeld MD, Peterson RE. 2,3,7,8-Tetrachlorodibenzo-p-dioxin-induced weight loss, a proposed mechanism. In: Tucker RE, Yong AL, Gray AP, eds. Human and environmental risks of chlorinated dioxins and related compounds. New York: Plenum Press, 1981: 405-413.
  • 8Brewster DW, Bombick DW, and Matsumua F. Rabbit serum hypertriglyceridemia after adminatlon of 2, 3, 7, 8-Tetra-chlorodibenzo-p-dioxin(TCDD).J. Toxical. Environ. Health. 1988, 24: 495-507.
  • 9Bombick DW, Matsumua F, and Madhukar BV. TCDD causes redliction in the Iow density lipoprotein(LDL) receptor activities in the hepatic plasma membrane of the guinea pig and rat. Biochem. Biophys. Res. Commun. 1984, 118: 548-554.
  • 10Poland A, Kuntson JK. 2. 3, 7, 8- Tetrachlorodibenzo-p-dioxin and related haiogenated aromatic hydrocarbons of the mechanism of toxicity. Auun Rev Pharmacol Toxicol, 1982, 22: 517-554.

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