人乳头瘤病毒E6、E7蛋白在宫颈分化上皮中的致病机制
出处
《江西医药》
CAS
2004年第6期432-434,共3页
Jiangxi Medical Journal
参考文献16
-
1Walboomers JM, Jacobs MV,Manos MM,et al. Human papillomavirus is a necessary cause of invasive cervical cancer worldwide. J Pathol,1999,189:12
-
2Zur Hausen H. Papillomaviruses and cancer from basic studies to clinical application. Nat Rev Cancer ,2002,2:342
-
3Zimmermann, Degenkolbe HR, Bernard HU, et al. The human papillomavirus type 16 E6 oncoprotein can down-regulate p53 activity by targeting the transcriptional coactivator CBP/p300. J Virol, 1999,73:6209
-
4Pim D, Storey A, Thomas M, et al. Mutational analysis of HPV-18 E6identifies domains required for p53 degradation in vitro, abolition of p53 transactivation in vivo and immortalisation of primary BMK cells.Oncogene, 1994,9:1869
-
5Lee, SS, Glaunsinger B, Mantovani F, et al. Multi-PDZ domain protein MUPP1 is a cellular target for both adenovirus E4- ORF1 and high-risk papillomavirus type 18 E6 oncoproteins, J Virol, 2000,74:9680
-
6Nguyen ML, Nguyen MM., Lee D, et al.The PDZ ligand domain of the human papillomavirus type 16 E6 protein is required for E6's induction of epithelial hyperplasia in vivo. J Virol, 2003, 77:6957
-
7Riley RR, Duensing S, Brake T, et al. Dissection of human papillomavirus E6 and E7 function in transgenic mouse models of cervical carcinogenesis. Cancer Res, 2003, 63:4862
-
8Nakamura TM, Morin GB, Chapman KB, et al. Telomerase catalytic subunit homologs from fission yeast and human. Science, 1997, 277:955
-
9Kyo S, Laimins LA. Telomerase activation by human papillomavirus type 16 E6 protein: induction of human telomerase reverse transcriptase expression through Myc and GC-rich Spl binding sites. J Virol,2001, 75:5559
-
10Veldman T, X Liu, H Yuan, et al. Human papillomavirus E6 and Myc proteins associate in vivo and bind to and cooperatively activate the telomerase reverse transcriptase promoter. Proc. Natl Acad Sci USA, 2003,100:8211
-
1阮怀珍,李希成,黎海蒂,赵邦云,蔡文琴,张遐.P物质参与伤害性刺激诱发的脊髓c-fos表达[J].科学通报,1994,39(13):1237-1240. 被引量：11
-
2陈守真,夏和霞,张炜.人乳头瘤病毒与宫颈癌[J].上海医药,2016,37(11):3-7. 被引量：5
-
3研究揭示致癌蛋白作用机理[J].现代临床医学生物工程学杂志,2003,9(2):122-122.
-
4郭建新,李力.HPV与宫颈癌疫苗研究进展[J].国外医学（妇幼保健分册）,2004,15(6):395-398. 被引量：9
-
5韩莉莉,刘晓婉,昆多孜.维吾尔族、汉族宫颈病变组织中HPV16E6、E7蛋白表达研究[J].中国社区医师,2017,33(11):97-98. 被引量：1
-
6刘小奕(综述),黄勇(审校).宫颈癌相关抑癌基因及其与人乳头瘤病毒的关系[J].国际肿瘤学杂志,2009,36(10):785-788. 被引量：2
-
7王琳,韩莉莉,玛依努尔.尼牙孜,隋霜.人乳头状瘤病毒E6、E7蛋白在维吾尔族及汉族宫颈癌发展中的表达及意义[J].中国医药导报,2015,12(21):75-78. 被引量：10
-
8王和平.Bcl—2 C—myc和P^53致癌蛋白的共同表达对评估大肠癌预后的意义[J].中国肛肠病杂志,2001,21(8):6-8.
-
9张遐,鞠躬.白细胞介素—1对大鼠下丘脑室旁核Fos癌蛋白和CRF的诱导作用[J].神经解剖学杂志,1991,7(2):169-172. 被引量：5
-
10曹俊,章月星,张志愿.人乳头状瘤病毒致口腔鳞癌发生的研究进展[J].上海口腔医学,2001,10(3):274-275.
