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结核分枝杆菌持续感染期分子机制的研究进展 被引量:1

Advance in the study of the molecular mechanism of Mycbacoterium
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出处 《中华结核和呼吸杂志》 CAS CSCD 北大核心 2005年第1期47-49,共3页 Chinese Journal of Tuberculosis and Respiratory Diseases
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  • 1Weber I, Fritz C, Ruttkowski S, et al. Anaerobic nitrate reductase (narGHJI) activity of Mycobacterium bovis BCG in vitro and its contribution to virulence in immunodeficient mice. Mol Microbiol,2000, 35 : 1017-1025.
  • 2Chemens DL, Lee BY, Howitz MA. Deviant expression of Rab5 on phagosomes containing the intracellular pathogens Mycobacterium tuberculosis and LegioneUa pneumophila is associated with altered phagosomal fate. Infect Immun, 2000, 68: 2671-2684.
  • 3Pieters J, Gatfield J. Hijacking the host: survival of pathogenic mycobacteria inside macrophages. Trend Microl,2002,10 : 142-146.
  • 4Vergne I, Chua J, Deretic V, et al. Tuberculosis Toxin Blocking Phagosome Maturation Inhibits a Novel Ca^2+/Camodulin-PI3K Hvps34 Cascade. J Exp Med, 2003, 198: 653-659.
  • 5McCune RM, Tompsrtt R, McDermott W. The fate of Mycobacterium tuberculosis in mouse tissues as determined by the microbial enumeration technique. Ⅱ. The conversion of tuberculous infection to the latent state by the administration of pyrazinamide and a companion drug. J Exp Med, 1957, 104: 763-802.
  • 6Wayne LG, Sohaskey CD. Nonreplicating persistence of Mycobacterium tuberculosis. Annu Rev Microbiol, 2001, 55: 139-163.
  • 7Scanga CA, Mohan VP, Joseph H, et al. Reactivation of latent tuberculosis: variations on the Comell murine model. Infect Immun,1999, 67: 4531-4538.
  • 8Betts JC, Lukey PT, Robb LC, et al. Evaluation of a nutrient starvation model of Mycobacterium tuberculosis persistence by gene and protein expression profiling. Mol Microbiol, 2002, 43: 717-731.
  • 9Cole ST, Brcsch R, Parkhill J, et al. Deciphering the biology of Mycobacterium tuberculosis from the complete genome sequence.Nature, 1998, 393: 537-544.
  • 10McKinney JD, Honer zu Bentrup K, Munoz-Elias E J, et al. Persistence of Mycobactefium tuberculosis in macrophages and mice requires the glyoxylate shunt enzyme isoxitrate lyase. Nature, 2000,406: 735-738.

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  • 1Ni Cheallaigh C, Keane J, l,avelle EC, et al. Aulophagy in the immune response to tuberc uh s s: clinical perspectives. Clin Exp lmmunol, 2011, 164: 291-300.
  • 2Thwaites GE, Chau TF, Stepniewska K, et al. Diagnosis of adult tuberculous meningitis by use of clinical and laboratory features. l,ancet. 2002, 360 : 1287-1292.
  • 3Jeffery K J, Read SJ, Peto TE, et al. Diagnosis of viral infections of the eentral nervous system clinical inteli)retation of PCR resuhs, l,aneet, 1997, 349: 313-317.
  • 4Deretic V, Singh S, Master S, et al. Myeobacterium tuberculosis inhibition of phagolysosome biogenesis and autophagy as a host defenee mechanism. Cell Microbiol, 2006, 8: 719-727.
  • 5Gulierrez MG, Master SS, Singh SB, et al. Autophagy is a defense mechanism inhibiting BCG and Myeobacterium tuberculosis survival in infecled macrophages. Cell, 2004, 119: 753-766.
  • 6Galimi R. Fxtrapuhnonary tuberculosis: tuberculous meningitis new developments. Eur Rev Med Pharmacol Sci, 2011, 15: 365- 386.
  • 7Garg RK. Tuberculous meningitis. Acta Neurol Stand, 2010, 122 : 75-90.
  • 8Rock RB, Olin M, Baker CA, el al. Central nervous system tuberculosis: pathogenesis and clinical aspects. Clin Mictx~biol Rev, 2008, 21: 243-261.

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