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SARS发病机制研究进展 被引量:1

Study improvement of pathogenesis of SARS
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摘要 严重急性呼吸综合征 (SARS)是由SARS病毒 (SARS_CoV)感染引起的 ,血管紧张素转化酶 2是SARS_CoV的功能性受体。SARS引起肺部病变的机制包括 :感染后所产生的原发性和多种继发性损伤 ;前炎症细胞因子的释放导致细胞因子调节障碍 ,产生吞噬红细胞作用 ;超敏反应与T细胞免疫失衡。
作者 严冬 李兰娟
出处 《国外医学(流行病学.传染病学分册)》 2005年第1期16-18,共3页 Epidemiology Lemology Foreign Medical Sciences
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参考文献17

  • 1郎振为,张立洁,张世杰,孟忻,李俊强,宋晨朝,孙琳,周育森.严重急性呼吸综合征3例尸检病理分析[J].中华病理学杂志,2003,32(3):201-204. 被引量:61
  • 2王福生,徐东平.SARS冠状病毒的特点和致病机制的研究[J].传染病信息,2003,16(2):67-68. 被引量:13
  • 3芮伟,张其鹏,石磊,卢铭.SARS冠状病毒基因组、蛋白质与侵入宿主细胞过程的研究近况[J].中华医学杂志,2003,83(11):913-921. 被引量:28
  • 4赵景民,周光德.传染性非典型肺炎的病理研究近况及展望[J].传染病信息,2003,16(2):69-70. 被引量:10
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二级参考文献16

  • 1WHO. WHO issues a global alert about cases of atypical pneumonia. (http://www. who. int/csr/sarsarchive/2003 -03 -12/en/).
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