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感染性休克的心肌损伤 被引量:1

Myocardial injury of septic shock
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出处 《中国感染控制杂志》 CAS 2005年第1期88-90,8,共4页 Chinese Journal of Infection Control
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参考文献36

  • 1吴文溪,吴肇光.外科基本问题──休克(3):低血容量性休克和感染性休克[J].外科理论与实践,2001,6(1). 被引量:3
  • 2许夕海,施光峰.感染性休克中脂多糖的信号传导机制研究进展[J].中国抗感染化疗杂志,2003,3(1):47-50. 被引量:5
  • 3Thiru, Y, Pathan N, Bignall S, et al. A myocardial cytotoxic process is involved in the cardiac dysfunction of meningococcal septic shock[J]. Crit Care Med, 2000, 28: 2979-2983.
  • 4Arlati S, Brenna S, Prencipe L, et al. Myocardial necrosis in ICU patients with acute non-cardiac disease: a prospective study[J]. Intensive Care Med, 2000, 26: 31-37.
  • 5Turner A, Tssamitros M, Bellomo R. Myocardial cell injury in septic shock[J]. Crit Care Med, 1999, 27: 1775-1780.
  • 6Ingalls R, Heine H, Linen E, et al. Lipopolysaccharide recognition CD14 and lipopolysaccharide receptors[J]. Infect Dis Clin North Am, 1999, 13: 341-353.
  • 7Katz Z, Chen K, Chen S, et al. Potent CD14 mediated signalling of human leukocytes by Escherichia coli can be mediated by interaction of whole bacteria and host cells without extensive prior release of endotoxin[J]. Immun, 1996, 64: 3592-3600.
  • 8Triantafilou M, Triantafilou K, Fernandez N. Rough and smooth forms of fluorescein-labelled bacterial endotoxin exhibit CD14/LBP dependent and independent binding that is influenced by endotoxin concentration[J]. Eur J Biochem, 2000, 267: 2218-2226.
  • 9Hamann L, Schumann R, Flad H, et al. Binding of lipopolysaccharide (LPS) to CHO cells does not correlate with LPS-induced NF-kappaB activation[J]. Eur J Immunnol, 2000, 30: 211-216.
  • 10Hamann L, Stamme C, Ulmer A J. Inhibition of LPS-induced activation of alveolar macrophages by high concentrations of LPS-binding protein[J]. Biochem Biophys Res Commun, 2002, 295(2): 553-560.

二级参考文献22

  • 1[1]Rietschel ET, Brade L, Lindner B, et al. Biochemistry of lipopo~lysaccharides. IN: Morrison DC, Ryan JL Eds: Bacterial endotoxic lipopolysaccharides[M]. Vol I. BocaRaton(USA):CRC Press,1994:56-57
  • 2[2]Ingalls RR, Heine H, Lien E, et al. Lipopolysaccharide recognition CD14 and lipopolysaccharide receptors[J]. Infect Dis Clin North Am,1999,13: 341-353
  • 3[3]Fearns C, Loskutoff DJ. Role of tumor necrosis factor alpha in induction of murine CD14 gene expression by lipopolysaccharide[J]. Infect Immunol,1997,65:4822-4831
  • 4[4]Gupta D, Kirkland TN, Viriyakosol S, et al. CD14 is a cell-activating receptor for bacterial peptidogkycan[J]. J Biolog Chem, 1996,271:23310-23316
  • 5[5]Katz SS, Chen K, Chen S, et al. Potent CD14-mediated signalling of human leukocytes by Escherichia coli can be mediated by interaction of whole bacteria and host cells without extensive prior release of endotoxin[J]. Immun, 1996,64:3592-3600
  • 6[6]Shapiro RA, Cunningham MD, Ratcliffe K, et al. Identification of CD14 residues involved in specific lipopolysaccharide recognition[J]. Imfect Immun, 1997,65: 293-297
  • 7[7]Troelstra A, Antal-Szalmas P, de Graaf-Miltenburg LA, et al. Saturable CD14-dependent binding of fluorescein-labeled lipopolysaccharide to human monocytes[J]. Infect Immun, 1997,65:2272-2277
  • 8[8]Tobias PS, Soldau K, Ulevitch RJ. Isolation of a lipopoly-saccharide-binding acute phase reactant from rabbit serum[J]. J exp Med, 1986, 164: 777-793
  • 9[9]Triantafilou M, Triantafilou K, Femandez N. Rough and smooth forms of fluorescein-labelled bacterial endotoxin exhibit CD14 /LBP dependent and independent binding that is influenced by endotoxin concentration[J]. Eur J Biochem, 2000,267: 2218-2226
  • 10[10]Hamann L, Schumann RR, Flad HD, et al. Binding of lipopoly-saccharide(LPS)to CHO cells does not correlate with LPS-induced. NF-kappa B activation[J]. Eur J Immunnol, 2000,30: 211-216

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