摘要
蜜环菌通过猪苓菌核表皮侵染菌核时,菌核表皮下层的菌丝具特异的拮抗反应,如细胞中有结晶颗粒出现,厚壁菌丝形成,部分薄壁菌丝有质壁分离现象。蜜环菌的侵染诱导了菌核防御结构的发生:离侵染点一定距离的部位出现由少量木质化菌丝和厚壁菌丝形成的疏松带状;蜜环菌侵入后,上述菌丝增多并紧密排列形成菌核的初级隔离腔,入侵的蜜环菌和部分菌核被隔离在腔中;在初级隔离腔形成的同时,外围的次级隔离腔开始发育。蜜环菌菌索和皮层菌丝分枝可突破初、次级隔离腔的壁,再以菌索产生的侵染带侵染菌核较外部的最后防线即三级隔离腔。本文较系统的阐述了蜜环菌侵染后菌核各防御结构的发生特点及功能。
The infection of Armillariella mellea could induce the genesis of defensee structure of the sclerotia of Grifola umbellata,such as the appearance of crystals in sclerotium cells,the occurrence of thick-walled hyphae,and the pla- smolysis at the hypodermal layer of sclerotia.A few thick-walled hyphae appear in the sclerotia as the rhizomorph of A.mellea adheres to the sclerotium epidermis, and then they increase in number and are closely arranged to form the wall of pri- mary isolation cavity.The invader together with part of sclerotia are enclosed in the cavity.The secondary isolation cavity subsequently develops in the outside of the primary one.The rhizomorph and cortical hyphae of A.mellea could break through the walls of primary and secondary or even third isolation cavities.The character and function of defense structure of sclerotia are discussed in this paper.
出处
《真菌学报》
CSCD
北大核心
1993年第4期283-288,共6页
基金
卫生部重点科研项目
关键词
猪苓
菌核
蜜环菌
防御结构
Grifola umbellata
Sclerotium
Armillariella mellea
Defense structure
