摘要
采用组织化学方法，结合超微结构观察探讨了硝苯吡啶对家兔缺血／再灌心肌细胞的作用。结果表明：硝苯吡啶对缺血１ｈ，再灌流３Ｏｍｉｎ的心肌细胞具有保护作用，但此受到保护的心肌细胞尚不能耐受继续再灌流的进一步损害，白细胞浸润可能是造成这种损害的原因。
ffects of nifedipine on the myocardisl cell of ischemia-reperfusion in rabbits were
ob-served with a histochemical metbd and dectromicroscopy,Results showed that nifedipinewas
of benefit to the myocardial cells with 30 min of reperfusion after l h of ischemia,but
thefavorable effect disappeared following the prolonged reperfusion. Leucocytes may induce
theinjury of the preserved myocardial cells.
出处
《湖南医科大学学报》
CSCD
1994年第1期29-32,共4页
Bulletin of Hunan Medical University
关键词
再灌注损伤
心肌缺血
硝苯吡啶
reperfusion
injury
myocardium
nifedipine
ultrastructure
histocy-tochemistry
succinate
dehydrogeriase
disease models,animal
rabbits
