摘要
对25例慢性硬膜下血肿的成份分析和结构观察,结果显示:血肿液纤维蛋白降解产物增高,。血肿液中新鲜红细胞,皱缩红细胞和隐形红细胞并存。血肿外壁为与硬脑膜紧密粘连的炎性结统组织,其内侧面有出血征。作者认为:CSDH的发病机理系头伤等病因造成患者硬膜下原发血肿,随血肿形成而继发纤溶亢进、凝血障碍。导致小血管渗血和新生不健全的炎性组织出血,如此反复不断的出血使血肿逐渐扩大,直至出现临床症状。
The composition and structure of 25 cases of chronic subdural hematoma(CSDH) were studied. The findings showed that the amount of fibrinogendegradation product was greatly elevated in the hematoma feuid, and there wasfresh, crenate and recessive red blood copps simultaneously. The capsule of CSDH,adhered tightly to the dura mater, was lined by inflammatory connective tissue withevidence of Bleeding in its inner side. Authors suggested that the pathogenesis ofCSDH was head injury and other causes formed primary subduralhematoma. Following the formation of hematoma, the hyp?brmolysis and thefailure of blool coagulation occured and which made oozing and bleeding from thesmall vessels and the new-forming imperfect inflammatory tissue. As this goes onrepeatedly. Continuous fresh bleeding made the hematoma bigger and biggergradually, until the clinical symptoms appeared.
