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心房重构在房颤的发生和维持中作用机制的研究进展 被引量:1

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机构地区 台州医院 浙江医院
出处 《心脑血管病防治》 2005年第2期56-58,共3页 CARDIO-CEREBROVASCULAR DISEASE PREVENTION AND TREATMENT
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参考文献22

  • 1Wijffels MC, Kirchhof CJ, Dorland R, et al. Atrial fibrillation begets atrial fibrillation. A study in awake chronically instrumented goats. Circulation, 1995, 92(7):1954-68.
  • 2Wang Z, Feng J, Shi H. Potential molecular basis of different physiological properties of the transient outward K+ current in rabbit and human atrial myocytes. Circ Res, 1999, 84(5):551-61.
  • 3Yue L, Melnyk P, Gaspo R. Molecular mechanisms underlying ionic remodeling in a dog model of atrial fibrllation. Circ Res, 1999, 84(7):776-84.
  • 4Grammer JB, Bosch RF, Kuhlkamp V, et al. Molecualr remodeling of Kv 4.3 potassium channels in human atrial fibrillation. J Cardiovasc Electrophysiol, 2000, 11(6): 626-33.
  • 5许春萱,张建成,黄从新,邓玉莲,施作霖,沈晓丽.心房颤动患者瞬间外向钾电流重构的分子基础[J].中华心律失常学杂志,2002,6(4):228-232. 被引量:10
  • 6Brundel BJ, Van Gelder IC, Henning RH, et al. Alterations in potassium channel gene expression in atria of patients with persistent and paroxysmal atrial fibrillation differential regulation of protein and mRNA levels for K+-channels. J Am Coll Cardiol, 2001,37(3):926-32.
  • 7Bosch RF, Zeng X, Grammer YB, et al. Ionic mechanisms of electrical remodeling in human atrial fibrillation. Cardiovasc Res, 1999,44(1):121-31.
  • 8张建成,黄从新,邓玉莲,许春萱,林立芳,胡锡衷.心房颤动患者离子重构的分子基础[J].中华心律失常学杂志,2002,6(2):84-88. 被引量:16
  • 9Ausma J, Dispersyn GD, Duimel H, et al. Changes in ultrastructural calcium distribution in goat atria during atrial fibrillation. J Mol Cell Cardiol, 2000,32(3):355-64.
  • 10Ohkusa T, Ueyama T, Yamada, et al. Alterations in cardiac sarcoplasmic reticulum Ca2+ regulatory proteins in the atrial tissue of patients with chronic atrial fibrillation. J Am Coll Cardiol, 1999,34(1):255-63.

二级参考文献33

  • 1Wijffels MC,Kirchhof CJ,Dorland R,et al.Atrial fibrillation begets Atrial fibrillation.A study in awake chronically instrumented goats.Circulation,1995,92:1954-1968.
  • 2Daoud EG,Bogun G,Goyal R,et al.Effect of AF on atrial refractoriness in humans.Circulation,1996,94:1600-1668.
  • 3Yue L,Feng J,Gaspo R,et al.Ionic remodding underlying action potential changes in a canine model of atrial fibrillation.Circ Res.1997,81:512-525.
  • 4Bosch RF,Zeng X,Grammer YB,et al.Ionic mechanisms of electrical remodeling in human atrial fibrillation.Cardiovasc Res,1999,44:121-131.
  • 5Van Wagoner DR,Pond AL,Lamorgese M,et al.Atrial L-type Ca2+ currents and human atrial fibrillation.Circ Res,1999,85:428-436.
  • 6Yue L,Melnyk P,Gaspo R,et al.Molecular mechanisms underlying ionic remodeling in a dog model of atrial fibrillation.Circ Res,1999,84:776-784.
  • 7Lai LP,Su MJ,Lin JL,et al.Down-regulation of L-type calcium channel and sarcopasmic reticular Ca2+ -ATPase mRNA in Human atrial fibrillation without significant change in the mRNA of ryanocine receptor,calsequestrin and phospholamban-an insight into the mechanism of atrial electrical remodeling.JACC,1999,33:1231-1237.
  • 8Brundel BJJM,Van Gelder IC,Henning RH,et al.Gene expression of proteins influencing calcium homeostasis in patients with persistent and paroxysmal atrial fibrillation.Cardiovas Res,1999,42:443-454.
  • 9Cheng TH,Lee FY,Wei J,et al.Comparison of calciumcurrent in isolated atrial myocytes from failing and nonfailing human hearts.Mol Cell Biochem,1996,157:157-162.
  • 10Boixel C,Gonzalez W,Louedec L,et al.Mechanisms of L-type Ca2+ current downregulation in rat atrial myocytes during heart failure.Circ Res,2001,89:607-613.

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二级引证文献6

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