1[1]Verma S,Wang CH,Li SH,et al. A self-fulfilling prophecy: C-reactive protein attenuates nitric oxide production and inhibits angiogenesis.[J].Circulation,2002, 106:913-919
2[2]Venugopal SK, Devaraj S,Yuhanna I,et al. Demonstration that Creactive protein decreases eNOS expression and bioactivity in human aortic endothelial cells. [J].Circulation,2002,106: 1439-1441[3].Verma S, Li SH, Badiwala MV, et al. Endothelial antagonism and interleukin-6 inhibition attenuate the proatherogenic effects of Creactive protein. [J ].Circulation, 2002,105: 1890-1896
3[4]Pasceri V, Willerson JT, Yeh ET,et al. Direct proinflammatory effect of C-reactive protein on human endothelial cells. [J]. Circulation.2002, 102: 2165-2168
4[5]Pasceri V, Cheng JS,Willerson JT,et al. Modulation of C-reactive protein-mediated'monocyte chemoattractant protein-1 induction in human endothelial cells by anti-atherosclerosis drugs. [J].Circulation.2001, 103:2531-2534
5[6]Torzewski M, Rist C, Mortensen RF, et al. C-reactive protein in the arterial intima: role of C-reactive protein receptor -dependent monocyte recruitment in atherogenesis. [J].Arterioscler Thromb Vasc Biol, 2000; 20: 2094-2099
6[7]Chang MK, Binder CJ,Torzewski M, et al. From the cover: C-reactive protein binds to both oxidized LDL and apoptotic cells through recognition of a common ligand :phosphorylcholine of oxidized phospholipids. [J]. Proc Natl Acad Sci USA,2002,99:13043-13048
7[8]Zwaka, TP, Hombach V, Torzewski M. C-reactive protein-mediated low density lipoprotein uptake by macrophages: implications for atherosclerosis. [J]. Circulation,2001; 103:1194-1197
8[9]Wang CH, Li SH, Weisel RD, et al. C-reactive protein upregulates angiotensin type 1 receptors in vascular smooth muscle cell.[J]. Criclation,. 2003,107:1783-1790
9[10]Hattori Y, Matsumura M, Kasai K. Vascular smooth muscle cell activation by C-reactive protein. [J].Cardiovasc Res,2003, 58:186-195
10[11]Williams TN, Zhang CX, Game BA,et al. C-reactive protein stimulates MMP-1 expression in U937 histiocytes through FcγRII and extracellular signal-regulated kinase pathway: an implication of CRP involvement in plaque destabilization. [J].Arterioscler Thromb Vasc Biol,2004,24:61-66
