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PDTC对人肺腺癌A549细胞COX-2、VEGF表达的影响及意义 被引量:2

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出处 《山东医药》 CAS 北大核心 2005年第17期24-25,共2页 Shandong Medical Journal
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参考文献2

  • 1Aggarwal BB.Nuclear factor-kappaB:the enemy within.Cancer Cell,2004,6:203~208.
  • 2Dormond O,Ruegg C.Regulation of endothelial cell integrin function and angiogenesis by COX-2,cAMP and Protein Kinase A.Thromb Haemost,2003,90:577~585.

同被引文献31

  • 1郑雪梅,张端莲,贺红旗,余瑛,杨勇.Caspase-3在肝癌组织中的表达及意义[J].数理医药学杂志,2005,18(5):428-429. 被引量:5
  • 2Brummel kamp TR, Ni jman SM, Dirac AM, et al. Loss of the cylin dromatosis tumor suppressor inhibits apoptosis by activating NF-kappa B[J]. Nature,2003,424:738-739.
  • 3HuaZou.Ahuman. Protein homologous to C. Elegonsced-4, partici- Pates in cytoehrome -c-dePendent,activation of caspase-3 Cell [J]. Med Pediatr Oneol, 2006,90:405 -413.
  • 4Ilmore TD. The Rel / nf-kappa B / I kappa B signal tmnsduction path- way and cancer[J]. Cancer Treat Res, 2006,115:241-265.
  • 5Chaud hary, P.M, M.T. Eby, A. Jasmin, et al. Activation of the NF-kappaB pathway by caspase-3 and its homologs [J]. Oncogene, 2005,19:4451-4460.
  • 6Gruker BM. Trcmscription factor NF-kB: a new perspective in anti- cancer therapy[J]. Postepy Biochem, 2004.50(2): 118-130.
  • 7Suh Rabson A.B.NF-kB activation in human prostate cancer:important mediator or epiphenomenon? [J]. J cell Biochem, 2004, 91 (1): 100-117.
  • 8Alexander Golks, Dirk Brenner, Peter H. Krammer, and Inna N. Lavrik, The selective inhibit ofNF-kB (PDTC)induces NF-κ B acti- vation[J].Cancer Res, 2006,15:245-248.
  • 9Jonsson G, Panlie S, Grandien A, et al. High level of caspase-3 corre- lates with resistance to death receptor induced apoptosis in bladder carcinoma cells [J]. Anticancer Res, 2005,23 (2):1213-1218.
  • 10Persad R, Liu C, Wu TT, et al. Over expression of caspase-3 in hepa- to cellular carcinomas[J]. Modem Pathology, 2004,17:861-867.

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