摘要
以叉鞭金藻为材料进行了有机磷农药——久效磷的毒性实验.结果表明,在久效磷胁迫下,微藻的生长受到了严重地抑制(72hEC_(50)= 3.49mg/L);其过氧化物酶(POD)活性降低,膜脂过氧化产物丙二醛(MDA)含量和电解质外渗率同步增加;超氧化物歧化酶(SOD)活性时而上升,时而下降.培养液中维生素C(V_tC)和维生素E(V_tE)的加入在一定程度上可缓解藻细胞的伤害.而核黄素(维生素B_2,简称V_tB_2)的加入可加重藻细胞的伤害.前者使电解质外渗率降低,后者则使电解质外渗率增加.显然,自由基参与了久效磷对细胞的伤害;而POD比SOD在清除叉鞭金藻细胞自由基中起着重要的作用.初步认为细胞的自由基伤害是久效磷抑制叉鞭金藻生长的原因之一.
The paper reported toxicity effect of monocrotophos on Dicrateria sp. , Uner monocrotophos stress, the activity of peroxidase (POD) declined; the level of lipid peroxidation (MDA content) and membrane permeability increased synchronously; the activity of superoxide dismutase (SOD) fluctuated irregularly. Vitamine C and vitamine B could alleviated the damage of cells,while vitamine B would exacerbate the damage . It is obvious that free-radical participated the damage of monocrotophos stress. The growth inhibition of Dicrateria sp. might be a result of free-radical damage.
出处
《海洋通报》
CSCD
北大核心
1995年第2期29-34,共6页
Marine Science Bulletin
基金
山东省自然科学基金资助项目
