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羊水中葡萄糖含量与宫内感染的关系 被引量:1

The relation between the glucose level in amniotic flu-id and intrauterine infection.
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摘要 本文测定62例足月孕产妇血和羊水中葡萄糖含量、羊水细菌培养、C反应蛋白及白细胞计数和分类,了解羊水中葡萄糖含量与宫内感染的关系。结果表明:在血糖浓度一致的惰况下,宫内感染组羊水中葡萄糖浓度(0.650±0.27mmol/L)明显低于无宫内感染组(1.549±0.63mmol/L).差异有极显著性,提示羊水中葡萄糖低水平与宫内感染有关。13例宫内感染,羊水中葡萄糖含量全部≤0.9mmol/L,正常组49例仅有8例≤0.9mmol/L,其诊断宫内感染的敏感性为61.9%,特异性为100%。羊水中葡萄糖测定诊断宫内感染优于C反应蛋白及白细胞计数.是一种廉价、快速、可推广的好方法。 he glucose levels in amniotic fluid and maternalblood,the amniotic fluid bacteria culture,the CRP andABC were measured in 62 term pregnant women. Theresults showed:Under the condition of that the bloodglucose levels were the same,the glucose level in amniotic fluid of intrauterine infection group (x = 0. 650±0. 27mmol/L)was obviously lower than that of non-intrauterine infection group (x = 1. 549 ± 0. 63mmol/L). The difference between the two groups was obvi-ous and suggested that the low glucose level in amni-otic fluid had been associated with the intrauterine in-fection. The glucose levels in amniotic fluid of all 13cases of intrauterine infection were < 0. 9mmol/L. A-mong the 49 cases of control group,there were & caseswhose glucose level in amniotic fluid were<0. 9mmol/L. The sensitivity of this method on diagnosing in-trauterine infection was 61. 9% and specifity was100%. The conclusion was that this method was better than the measuring of CRP and WBC for diagnos-ing the intrauterine infection.
出处 《中国实用妇科与产科杂志》 CAS CSCD 1995年第3期149-150,共2页 Chinese Journal of Practical Gynecology and Obstetrics
关键词 羊水 葡萄糖 宫内感染 病理学 Amniotic fluid GlucoseIntrauterin infection
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  • 1宗利丽,宋伯来,潘石蕾,王佳勇.孕妇血清甲胎蛋白值水平与早产低体重儿的关系[J].第一军医大学学报,1994,14(4):286-287. 被引量:14
  • 2Wenstrom KD, Andrews WW, Hauth JC, et al. Elevated secondtrimester amniotic fluid interleukin-6 levels predict preterm delivery. Am J Obstet Gynecol, 1998, 178:546 - 550.
  • 3Ghidini A, Jenkins CB, Spong CY, et al. Elevated amniotic fluid interleukin-6 levels during the early second trimester are associated with greater risk of subsequent preterm delivery.Am J Reprod lmmunol, 1997, 37:227 - 231.
  • 4Murtha AP, Greig PC, Jimmerson CE, et al. Matermal serum interleukin-6 concentration as amarker for impeding preterm delivery. Obstet Gynecol, 1998, 91 : 161 - 164.
  • 5Bahar AM, Ghalib HW, Moosa RA, et al.Maternal serym interleukin-6, interleukin-8, tumor necrosis factor-alpha and interferon-gamma in preterrn labor. Acta Obstct Gynecol Scand, 2003,82:543 - 549.
  • 6Ghezzi F, Franchi M, Raio L, et al. Elevated amniotie fluid C-reactive protein at the time of genetic amniocentesis is a marker for preterrn delivery. Am J Obstet Gynecol, 2002, 186:268 - 273.
  • 7Ozer KT, Kavak ZN, Gokaslan H, et al. Predictive power of maternal serum and amniotic fluid CRP and PAPP-A concentrations at the time of genetic amniocentesis for the preterm delivery. Eur J Obstet Gynecol Reprod Biol, 2005, 122:187 - 190.
  • 8Yoon BH, Oh SY, Romero R, et al. An elevated amniotic fluid matrix metalloproteinase-8 level at the time of mid-trimester genetic amniocentesis is a risk factor for spontaneous preterm delivery. Am J Obstet Gynecol, 2001, 185:1162 - 1167.
  • 9Stefos T, Sotiriadis A, Kaponis A, et al. Amniotic fluid glucose at the time of genetic amniocentesis: correlation with duration of pregnancy and birthweight.Eur J Obstet Gynecol Reprod Biol, 2003, 106:144- 147.
  • 10Ellis M J, Livesey JH, Inder WJ, et al. Plasma corticotropin-releasing hormone and unconjuated estriol in human prgnancy: Gestational patterns and ability to predict preterm delivery. Am J Obstet Gynecol, 2002, 186:94 - 99.

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