期刊文献+

短期小剂量硫唑嘌呤致粒细胞缺乏临床分析 被引量:3

下载PDF
导出
出处 《中国药物与临床》 CAS 2005年第8期619-620,共2页 Chinese Remedies & Clinics
  • 相关文献

参考文献11

二级参考文献46

  • 1Evans WE, McLeod HL. Pharmacogenomics-drug disposition, drug targets, and side effects. New Engl J Med, 2003,348: 538-549.
  • 2Roses AD. Pharmaeogenetics place in modem medical science and practice. Life Sci, 2002,70 : 1471-1480.
  • 3Chen Z, Karaplis AC, Ackerman SL, et al. Mice deficient in methylenetetrahydrofolate reductase exhibit hyperhomocysteinemia and decreased methylation capacity with neuropathology and aortic lipid deposition . Hum Mol Genet,2001,10:433-443.
  • 4Skibola CF, Smith MT, Kane E, et al. Polymorphisms in the methylenetetrahydrofolate reduetase gene are associated with susceptibility to acute leukemia in adults. Proc Natl Acad Sci USA, 1999,96:12810-12815.
  • 5van der Put NM, Gabreels F, Stevens EM, et al. A second common mutation in the methylenetetrahydrofolate recluctase gene: an additional risk factor for neural-tube defects. Am J Hum Genet, 1998,62 : 1044-1051.
  • 6Haagsma CJ, Blom HJ, van Riel PL, et al. Influence of sulphasalazine,methotrexate, and the combination of both on plasma homocysteine concentrations in patients with rheumatoid arthritis.Ann Rheum Dis, 1999,58:79-84.
  • 7van Ede AE, Laan RF, Blom HJ, et al. The C677T mutation in the methylenetetrahydrofolate reductase gene: a genetic risk factor for methotrexate-related elevation of liver enzymes in rheumatoid arthritis patients. Arthritis Rheum, 2001,44:2525-2530.
  • 8Urano W, Taniguchi A, Yamanaka H, et al. Polymorphisms in the methylenetetrahydrofolate reductase gene were associated with both the efficacy and the toxicity of methotrexate used for the treatment of rheumatoid arthritis, as evidenced by single locus and haplotype analyses. Pharmacogenetics, 2002,12:183-190.
  • 9Krynetsld EY, Evans WE. Pharmacogenetics as a molecular basis for individualized drug therapy: the thiopurine S-methyltransferase paradigm. Pharm Res, 1999,16: 342-349.
  • 10McLeod HL, Siva C. The thiopurine S-methyltransferase gene locus-implications for clinical pharmacogenomics .Pharmacogenomics,2002,3 : 89-98.

共引文献41

同被引文献19

  • 1杨程德,郭强,李挺,鲍春德,顾越英,陈顺乐.硫唑嘌呤致SLE骨髓抑制者的TPMT酶基因多态性[J].上海第二医科大学学报,2004,24(B11):67-69. 被引量:7
  • 2詹钟平,杨岫岩,黄民,王一西,连帆,梁柳琴,叶玉津,许韩师.硫唑嘌呤致血液系统危象的临床特征与酶学基础[J].中华风湿病学杂志,2005,9(3):145-148. 被引量:16
  • 3王文涛,严律南,李波,曾勇,文天夫,赵纪春,张鸣,马玉魁,李志辉,徐明清,杨加印.肝移植术后应用硫唑嘌呤致严重急性粒细胞缺乏二例[J].中华器官移植杂志,2005,26(12):712-712. 被引量:2
  • 4顾菊林,陈裕充,温海.顾菊林[J].临床皮肤科杂志,2006,35(3):165-165. 被引量:3
  • 5Fraser AG, Orchard TR, Jewell DP. The efficacy of azathioprine for the treatment of inflammatory bowel disease-a 30 year review. Gut, 2002, 50: 485-489.
  • 6Arnott ID, Watts D, Satsangi J. Azathioprine and anti-TNF alpha therapies in Crohn's disease: a review of pharmacology, clinical efficacy and safety. Pharmacol Res, 2003, 47: 1-10.
  • 7Hibi T, Naganuma M, Kitahora T, et al. Low-dose azathioprine is effective and safe for maintenance of remission in patients with ulcerative colitis. J Gastroenterol, 2003, 38: 740-746.
  • 8Gaffney K, Scott DGI. Azathioprine and cyclophosphamide in the treatment of rheumatoid arthritis. Br J Rheumatol, 1998, 37: 824- 836.
  • 9Willkens RF, Sharp JT, Stablein D, et al. Comparison of azathioprine, methotrexate, and the combination of the two in the treatment of rheumatoid arthritis. Arthritis Rheum, 1995, 38: 1799- 1806.
  • 10Naughton MA, Bataglia E, O'Brien S, et al. Identification of thiopurine methyltransferase (TPMT) polymorphisms cannot predict myelosuppression in systemic lupus erythematosus patients taking azathioprine. Rheumatology, 1999, 38: 640-644.

引证文献3

二级引证文献10

相关作者

内容加载中请稍等...

相关机构

内容加载中请稍等...

相关主题

内容加载中请稍等...

浏览历史

内容加载中请稍等...
;
使用帮助 返回顶部