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化学致癌的表遗传机制 被引量:1

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出处 《毒理学杂志》 CAS CSCD 北大核心 2005年第A03期176-177,共2页 Journal of Toxicology
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  • 1苏世标,杨鲁静,张卫,靳雅丽,聂继华,童建.某铀矿工人痰细胞中p16和MGMT基因的甲基化状态[J].中华劳动卫生职业病杂志,2006,24(2):92-95. 被引量:5
  • 2Moiseeva EP,Manson MM. Dietary chemopreventlve phytochemicals: too little or too much[J]. Cancer Prev Res,2009,2:611-616.
  • 3Klimczak A,Malinowska K,Kubiak K. Tumour illnesses and nutrition [J ]. Pol Merkur Lekarski, 2009,27 : 242-244.
  • 4Satia JA, Littman A,Slatore CG, et al. Long-term use of beta-carotene, retinol,lycopene,and lutein supplements and lung cancer risk: results from the VITamins and Lifestyle (VITAL) study[J]. Am J Epidemiol, 2009,169 : 815-828.
  • 5Stidley CA, Picchi MA, Leng S, et al. Multi-vitamins,folate,and green vegetables protect - against gene promoter methylation in the aerodigestive tract of smokers[ J ]. Cancer Res, 2010,70 : 568-574.
  • 6Vaissiere T, Hung RJ,Zaridze D,et al. Quantitative analysis of DNA methylation profiles in lung cancer identifies aberrant DNA methylation of specific genes and its association with gender and cancer risk factors [J ]. Cancer Res, 2009,69 : 243-252.
  • 7Adriana A,Max C. Epigenetics in metal carcinogenesis:nickel,arsenic, chromium and cadmium [ J ]. Metallomics, 2009,1 : 222-228.
  • 8Govindarajan B,Klafter R,Miller MS,et al. Reactive oxygen-induced carcinogenesis causes hypermethylation of p16(Ink4a) and activation of MAP kinase[ J]. Mol Med, 2002, g : 1-8.
  • 9Kowara R,Salnikow K,Diwan BA,et al. Reduced Fhit protein expression in nickel transformed mouse cells and in nickel-induced murine sarcomas[J]. Mol Cell Biochem, 2004,255: 195-202.
  • 10Ji W,Yang L,Yu L,et al. Epigenetic silencing of O6-methylguanine DNA methyltransferase gene in NiS-transformed cells[J]. Carcinogenesis, 2005,29 : 1267-1275.

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