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糖尿病肾病患者的处理 被引量:3

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出处 《中国全科医学》 CAS CSCD 2005年第17期1407-1407,共1页 Chinese General Practice
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  • 1贾汝汉,高苹.糖尿病肾病肾小管间质病变研究进展[J].中国中西医结合肾病杂志,2005,6(6):311-314. 被引量:18
  • 2邢淑丽,郑君芙,黄文政.单侧肾切除STZ诱导糖尿病肾病大鼠动物模型研究[J].中国中医急症,2006,15(6):643-644. 被引量:51
  • 3肖新怀,陈澍.拮抗转化生长因子β活性对糖尿病肾病的防治作用[J].中国药物与临床,2006,6(10):725-727. 被引量:1
  • 4Lappin DWP,Doran P,Godson C,et al.Gene responds to hypereaemia[J].EXP Nephrol,2002,10:120-129.
  • 5Gilbert RE,Cooper ME.The tubulointerstitiμm in progressive diabetic kidney disease:more than an aftermath of glomerular injury[J]? Kidney Int,1999,56:1627-1637.
  • 6Lan S,Vander Geest RN,Verhagen NA,et al.Seceetion of collogen type Ⅳ by hμman renal fibroblasts is increased by high glucose via a TGF-bata-independent pathway[J].Nephrol Dial Transplant,2004,19:1694-1701.
  • 7Sanai T,Sobka T,Johson T,et al.Exoression of cytoskeletal proteins during the course of experimental diabetic nephropathy[J].DIabetologia,2000,43:91-100.
  • 8Yokoi H,Suga Wara A,Mukoyama M,et al.Role of connective tissue growth factor in profibrotic action of transforming growth factor-beta:apotential target for preventing renal fibrosis[J].Am J Kidney,Dis,2001,38:134-138.
  • 9tan S, Vander Ceest RN, Verhagen NA, et al. Seceetion of collogen type 1V by human renal fibroblasts is increeased by high glucose via a TGFbata - independent pathway [ J ]. Nephrol Dial Transplant, 2004,19 : 1694.
  • 10王海燕主编.肾脏病学.2版.北京:人民卫生出版社,2001:1545.

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