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一种新的哮喘易感基因——ADAM33基因 被引量:4

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出处 《国外医学(呼吸系统分册)》 2005年第9期647-649,共3页 Section of Respiratory System Foreign Medical Sciences
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  • 1Cookson W. A new gene for asthma: would you ADAM and Eve it? Trends Genet, 2003,19(4): 169-172.
  • 2Van Eerdewegh P,Little RD, Dupuis J, et al. Association of the ADAM33 gene with asthma and bronchial hyperresponsiveness.Nature, 2002,418 (6896) :426-430.
  • 3De Sanctis GT, Merchant M, Beier DR, et al. Quantitative locus analysis of airway hyperresponsiveness in A/J and C57BL/6J mice. Nat Genet, 1995,11(2): 150-154.
  • 4Yoshinaka T, Nishii K, Yamada K, et al. Identification and characterization of novel mouse and human ADAM33s with potential metalloprotease activity. Gene, 2002, 282 ( 1-2 ): 227-236.
  • 5Gunn TM, Azarani A, Kim PH, et al. Identification and preliminary characterization of mouse Adam33. BMC Genet,2002,3(1) :2.
  • 6Primakoff P, Myles DG. The ADAM gene family: surface proteins with adhesion and protease activity. Trends Genet,2000,16(2) :83-87.
  • 7Stone AL, Kroeger M, Sang QX. Structure-function analysis of the ADAM family of disintegrin-like and metalloproteinasecontaining proteins. J Protein Chem, 1999,18(4) :447-465.
  • 8Shirakabe K, Wakatsuki S, Kurisaki T, et al. Roles of Meltrin beta /ADAM19 in the processing of neuregulin. J Biol Chem,2001,276(12) :9352-9358.
  • 9Cerretti DP. Characterization of the tumour necrosis factor alphaconverting enzyme, TACE/ADAM17. Biochem Soc Trans, 1999,27(2) :219-223.
  • 10Schlondorff J, Blobel CP. Metalloprotease-disintegrins: modular proteins capable of promoting cell-cell interactions and triggering signals by protein-ectodomain shedding. J Cell Sci, 1999,112 (Pt 21) :3603-3617.

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