1Tsujimoto Y, Finger LK, Yunis J,et al. Cloning of the chromesome breakpoint of neopastic B cells with the (14:18) chromosome translocation[J]. Science, 1984,226:1079.
2Campos L,Rouaul JP,Sabido O ,et al. High expression of Bcl-2 protein in acute myeloid leukemia cell is associated with poor response to Chemotherapy[J]. Blood, 1993,81:3091-3096.
3Oltvai ZN,Milliman CI,Korsmeyer SJ. Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that acelerates programmes cell death[J].Cell, 1993,74(4) :609-619.
4Hockenbery DM,Oltvai ZN, Yin XM,et al. Bcl-2 functions in a antixidant pathway to prevent apoptosis cell[J]. Cell, 1993,75:241-251.
5Bany G,Miyashita T, Williamson JR, et al. Aprptosis induced by withdrawal of interleukin-3(IL-3) from and IL-3-dependent hematopoietic cell line is associated with repartitioning of in tracellwlar calcium and is blocked by enforced Bcl-2 oncoprotein production[J]. J Biol Chem, 1993,268:6511.
6Smete LA,Bery JVD,Acton D,et al. Bcl-2 expression and mitochondrial activity in leukemic cells with different sensitivity to glucocorticoid induced apoptosis[J]. Blood, 1994,84:1613.
7Reed JC. Bcl-2:prevention of apoptosis as a mechanism of drug resistance[J]. Hematol oncol Clin North Am,1995,9(2):451-473.
8Rosse T, Olivier R,Monney L, et al. Bcl-2 Prolongs cell survival after Bax-induced release of cytochrome c[J]. Nature,1998,391:441-442.
9Meijerink JP,Smetsers TF, Sloetjes AW, et al. Bax mutations in cell lines derived from hematological malignancies[J]. Leukemia,1995,9(11):1828-1832.
10Sedlak TW,tVai ZN,Yang E,et al. Multiple Bcl-2 family members demonstrate selective dimerizations with Bax[J]. Proc Natl Acad Sci USA,1995,92(17) :7834-7838.
