1Randle PJ, Garland PB, Hales CN,et al.The glucose-fatty acid cycle: its role in insulin sensitivity and the metabolic disturbances of diabetes mellitus. Lancet,1963, 1: 785-789.
2Ginsberg HN. Basic mechanisms underlying the common hyper-triglyceridemia and low HDL cholesterol levels. Diabetes, 1996, 45: S27-S30.
3Egan BM, Stepniakowski KT. Evidence linking fatty acids, the risk factor cluster, and vascular pathophysiology. Implications for the diabetic hypertensive patient. In: Sowers JR(ed). Contemporary Endocrinology: Endocrinology of the vasculature. Totowa MJ: Humana Press Inc, 1996. 157-172.
5Grundy SM, Brewer B, Cleeman JI, et al. Definition of metabolic syndrome. Report of the National Heart, Lung and Blood Institute/American Heart Association conference on scientific issues related to definition. Circulation, 2004, 109: 433-438.
6Alerti KGMM, Zimmet PZ for the WHO Consultation. Definition, diagnosis and classification of diabetes mellitus. Provisional report of a WHO consultation. Diabetes Med, 1998, 15: 539-553.
7Hanson RL, imperatore G, Bennett PH, et al. Components of the "metabolic syndrome" and incidence of type 2 diabetes. Diabetes, 2002, 51: 3120-3127.
8Bolinder J, Kager L, Ostman J, et al. Differences at the receptor and postreceptor levels between human omental and subcutaneous adipose tissue in the action of insulin on lipolysis. Diabetes, 1983, 32: 117-123.
9Jensen MD, Haymond MW, Rizza RA, et al. Influence of body fat distribution on free fatty acid metabolism in obesity. J Clin Invest, 1989, 83: 1168-1173.
10Boden G, Jadali F, White J, et al. Effects of fat on insulin-stimulated carbohydrate metabolism in normal men. J Clin Invest, 1991, 88: 960-966.
9WHO/NUT/NCD.Obesity:preventing and managing the global epidemic.Report of a WHO consultation on obesity.Geneva:WHO,1997,2:3-5.
10Carianne L,G.Henk V,Eryn T L,et al.Comparison of methods to assess body fat in non-obese six to seven-year-old children.Clinical Nutrition,2010,29:317-322.
