1Haghighi K, Schmidt AG, Hoit BD, et al. Superinhibition of sarcoplasmic reticulum function by phospholamban induces cardiac contractile failure[J]. J Biol Chem,2001,276(26) :24145 -24152.
2Pollesello P, Annila A. Structure of the 1 - 36 N - terminal fragment of human phospholamban phosphorylated at Ser - 16 and Thr - 17[J]. Biophys J,2002, 83(1):484 -490.
3Kirby TL, Karim CB, Thomas DD. Electron paramagnetic resonance reveals a large - scale conformational change in the cytoplasmic domain of phospholamban upon binding to the sarcoplasmic reticulum Ca - ATPase [ J ]. Biochemistry, 2004, 43 ( 19 ): 5842 - 5852.
4Li J, Bigelow DJ, Squier TC. Conformational changes within the cytosolic portion of phospholamban upon release of Ca-ATPase inhibition [ J ]. Biochemistry,2004,43 ( 13 ) :3870 - 3879.
5Schmidt AG, Zhai J, Carr AN,et al. Structural and functional implications of the phospholamban hinge domain: impaired SR Ca^2 + uptake as a primary cause of heart failure[ J]. Cardiovasc Res ,2002,56(2) :248 -259.
7Song Q, Young KB, Chu G,et al. Overexpression of phospholamban in slow- twitch skeletal muscle is associated with depressed contractile function and muscle remodeling [ J ]. FASEB J, 2004, 18 ( 9 ):974 - 976.
9Bellinger AM, Mongillo M, Marks AR. Stressed out: the skeletal muscle ryanodine receptor as a target of stress [ J ]. J Clin Invest, 2008, 118(2) :445 -453.
10Samso M, Shen X, Allen PD. Structural characterization of the RyR1-FKBPI2 interaction[J]. J Mol Biol, 2006, 356(4) :917 - 927.
