1Devonald MA, Karet FE. Renal epithelial traffic jams and one-way streets. J Am Soc Nephrol, 2004, 15: 1370-1381.
2Gormley K, Dong Y, Sagnella GA.Regulation of the epithelial sodium channel by accessory proteins.Biochem J, 2003,371: 1-14.
3Shimkets RA, Warnock DG, Bositis CM,et al.Liddle's syndrome: Heritable human hypertension caused by mutations in the beta subunit of the epithelial sodium channel. Cell, 1994,79: 407-414.
4Pradervand S, Wang Q, Burnier M,et al.A mouse model for Liddle's syndrome. J Am Soc Nephrol, 1999,10: 2527-2533.
5Dahlmann A, Pradervand S, Hummler E,et al.Mineralocorticoid regulation of epithelial Na+ channels is maintained in a mouse model of Liddle's syndrome. Am J Physiol Renal Physiol, 2003,285: F310-F318.
6Nielsen S, Chou CL, Marples D,et al. Vasopressin increases water permeability of kidney collecting duct by inducing translocation of aquaporin-CD water channels to plasma membrane. Proc Natl Acad Sci USA,1995, 92: 1013-1017.
7Gustafson CE, Katsura T, McKee M,et al.Recycling of AQP2 occurs through a temperature- and bafilomycin-sensitive trans-Golgi-associated compartment. Am J Physiol Renal Physiol, 2000,278: F317-F326.
8Lu H, Sun TX, Bouley R,et al.Inhibition of endocytosis causes phosphorylation (S256)-independent plasma membrane accumulation of AQP-2. Am J Physiol Renal Physiol, 2003,286:F233-F243.
9Tan CM, Nickols HH, Limbird LE.Appropriate polarization following pharmacological rescue of V2 vasopressin receptors encoded by X-linked nephrogenic diabetes insipidus alleles involves a conformation of the receptor that also attains mature glycosylation. J Biol Chem, 2003,278: 35678-35686.
10Marr N, Bichet DG, Lonergan M,et al.Heteroligomerization of an Aquaporin-2 mutant with wild-type Aquaporin-2 and their misrouting to late endosomes/lysosomes explains dominant nephrogenic diabetes insipidus. Hum Mol Genet, 2002,11: 779-789.