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一氧化氮对心血管系统的调控 被引量：2

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摘要 冯清平教授从事NO对心血管系统调控的研究多年，曾在Circulation发表近20篇有关论文。冯教授现在已经成为加拿大心血管药理学专家，多年参与该国国家医药科研资助基金委员会。此文系冯教授他自己研究的NO对心血管系统影响的总结性文章。本刊特邀作稿以飨读者。冯教授系我刊外籍编委。
作者 向芙莉 冯清平
机构地区 西安大略大学医院内科
出处 《高血压杂志》 CSCD 北大核心 2005年第11期673-675,共3页 Chinese Journal of Hypertension
关键词 心血管系统 一氧化氮 调控 心血管药理学 科研资助 教授 加拿大 委员会
分类号 R54 [医药卫生—心血管疾病]
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参考文献17

  • 1Razavi IM, Hamilton JA, Feng Q. Modulation of apoptosis by nitric oxide: implications in myocardial ischemia and heart failure [J]. Pharm & Ther,2005,106:147-162.
  • 2Furchgott RF, Zawadzki JV. The obligatory role of endothelial cells in the relaxation of arterial smooth muscle by acetylcholine [J]. Nature, 1980,288: 373-376.
  • 3Palmer PM, Ferrige AG, Moncada S. Nitric oxide release accounts for the biological activity of endothelium-derived relaxing factor[J]. Nature, 1987,327:524-526.
  • 4Kojda G, Kottenberg K. Regulation of basal myocardial function by NO[J]. Cardiovasc Res, 1999,41: 514-523.
  • 5Kaasik A, Minajeva A, De Sousa E, et al. Nitric oxide inhibits cardiac energy production via inhibition of mitochondrial creatine kinase[J]. FEBS Lett, 1999,444: 75-77.
  • 6Sears CE, Bryant SM, Ashley EA, et al. Cardiac neuronal nitric oxide synthase isoform regulates myocardial contraction and calcium handling[J]. Cir Res, 2003,92: 52-59.
  • 7Jones SP, Girod WG, Palazzo AJ, et al. Myocardial ischemiareperfusion injury is exacerbated in absence of endothelial cell nitric oxide synthase[J]. Am J Physiol, 1999,276:1567-1573.
  • 8Kanno S, Lee PC, Zhang Y, et al. Attenuation of myocardial ischemia/reperfusion injury by superinduction of inducible nitric oxide synthase[J]. Circulation,2000,101:2742-2748.
  • 9Guo Y, Jones WK, Xuan YT, et al. The late phase of ischemic preconditioning is abrogated by targeted disruption of the inducible NO synthase gene[J]. Pro Natl Acad Sci USA, 1999, 96:11507-11512.
  • 10Rui T, Cepinskas G, Feng Q, et al. Delayed preconditioning in cardiac myocytes with respect to development of a proinflammatory phenotype: role of SOD and NOS[J]. Cardiovasc Res,2003,59:901-911.

同被引文献15

  • 1齐志敏,张莹,王倩.大鼠心肌缺血再灌注损伤实验模型研究[J].中国临床康复,2004,8(30):6620-6621. 被引量：66
  • 2仵文英,林秀云,郭琦,杨云,李艳玲.兔体内甘草酸锌和葡萄糖酸锌药动学比较[J].微量元素与健康研究,1994,11(3):4-5. 被引量：4
  • 3石增成,安丰双.一氧化氮与心肌缺血再灌注损伤研究进展[J].医学综述,1997,3(5):214-216. 被引量：3
  • 4Singh D,Chopra K.The effect of naringin,a bioflavonoid on ischemia-reperfusion induced renal injury in rats[J].Pharmacol Res,2004,50(2):187.
  • 5Gaur V,Aggarwal A,Kumar A.Protective effect of naringin against ischemic reperfusion cerebral injury:possible neurobehavioral,biochemical and cellular alterations in rat brain[J].Eur J Pharmacol,2009,616(1-3) :147.
  • 6Jones SP,Bolli R.The ubiquitous role of nitric oxide in cardioprotection[J].J Mol Cell Cardiol,2006,40(1):16.
  • 7Pozo Navas B,Stes sel H,Wolkart G,et al.Role of myocardial nitric oxide in diabetic ischemia-reperfusion dysfunction:studies in mice with myocyte-specific overexpression of endothelial nitric-oxide synthase[J].J Pharmacol Exp Ther,2006,319 (2):729.
  • 8Duranski MR,Elrod JW,Calvert JW,et al.Genetic Overexpression of eNOS Attenuates Hepatic ischemia-reperfusion injury[J].Am J Phys iol Heart Circ Physiol,2006,291 (6):H2980.
  • 9Pevni D,Frolkis I,Shapira I,et al.Ischaemia or reperfusion:which is main trigger for changes in nitric oxide mRNA synthases expression[J].Eur J Clin Invest,2005,35 (9):546.
  • 10Hu A,J iao X,Gao E,et al.Chronic beta-adrenergic receptor stimulation induces cardiac apoptosis and aggravates myocardial ischemia/reperfusion injury by provoking inducible nitric-oxide synthase-mediated nitrative stress[J].J Pharmacol Exp Ther,2006,318 (2):469.

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高血压杂志
2005年 第11期

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