1[1]Priori SG,Barhanin J,Hauer RNW,et al.Genetic and molecular basis of cardiac arrhythmias:impact on clinical management (Part Ⅰ and Ⅱ).Circulation,1999,99:518-528.
2[2]Nagatomo T,January CT,Ye B,et al.Rate-dependent QT shorting mechanism for the LQT3 △KPQ mutant.Cardiovascular Research,2002,54:624-629.
4[4]Priori SG,Napolitano C,Schwartz PJ.Low penetrance in the long-QT syndrome.Clinical impct.Circulation,1999,99:529-533.
5[5]Keating MT,Atkinson D,Dunn C,et al.Linkage of a cardiac arrhythmia,the long QT syndrome,and the Harvey ras-1 gene.Science,1991,252:704-706.
6[6]Neyroud N,Tesson F,Denjoy I,et al.A novel mutation on the potassium channel gene KVLQT1 causes the Jervell and Lang-Nielsen cardioauditory syndrome.Nat Genet,1997,15:186-189.
7[7]Barhanin J,Lesage F,Guillemare E,et al.KVLQT1 and IsK (mink) proteins associate to form the IKS cardiac potassium channel.Nature,1996,384:78-80.
8[8]Jiang C,Atkinson D,Towbin JA,et al.Two long QT syndrome loci map to chromosomes 3 and 7 with evidence for further heterogeneity.Nat Genet,1994,8:141-147.
9[9]Curran ME,Splawski I,Timothy KW,et al.A molecular basis for cardiac arrhythmia:HERG mutations cause long QT syndrome.Cell,1995,80:795-803.
