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心房颤动犬一氧化氮合成途径的变化:氧化应激的可能机制

Changes of nitric oxide synthesis pathway in the canine atrial fibrillation model;the possible mechanism for oridative stress
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摘要 一氧化氮(NO)代谢紊乱与许多心血管疾病,如动脉粥样硬化、高血压、心力衰竭和高脂血症有密切关系,但是NO在心房颤动(房颤)中的作用还不完全清楚.
出处 《中华心律失常学杂志》 2005年第6期452-453,共2页 Chinese Journal of Cardiac Arrhythmias
基金 黑龙江省青年科学技术专项资金(10551188)
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参考文献5

  • 1Korantzopoulos P, Kolettis T, Siogas K, et al. Atrial fibrillation and electrical remodeling: the potential role of inflammation and oxidative stress. Med Sci Monit,2003,9 : RA225-229.
  • 2Davis ME, Grumbach IM, Fukai T,et al. Shear stress regulates endothelial nitric-oxide synthase promoter activity through nuclear factor kappaB binding. J Biol Chem, 2004,279 : 163-168.
  • 3Sata N, Hamada N, Horinouchi T, et al. C-reactive protein and atrial fibrillation. Is inflammation a consequence or a cause of atrial fibrillation? Jpn Heart J ,2004,45:441-445.
  • 4Anderson JL, Allen Maycock CA, Lappe DL, et al. Frequency of elevation of C-reactive protein in atrial fibrillation. Am J Cardiol, 2004,94 : 1255-1259.
  • 5Calderone A. The therapeutic effect of natriuretic peptides in heart failure; differential regulation of endothelial and inducible nitric oxide synthases. Heart Fail Rev,2003 ,8 :55-70.

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